Shintani Yasunori | Osaka University

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著作論文

PJ-047 The Combined Therapy of Angiotensin Converting Enzyme Inhibitor, Angiotensin II Receptor Blocker and Beta-blockers Submaximally Limits Infarct Size(Acute Myocardial Infarction, Basic 2 (IHD) : PJ8)(Poster Session (Japanese))
PJ-046 Celiprolol, a Selective β_1-Adrenoceptor Antagonist, Limits Infarct Size via NO-dependent Mechanisms(Acute Myocardial Infarction, Basic 2 (IHD) : PJ8)(Poster Session (Japanese))
1 Evidence for Human Atrial Natriuretic Peptide and Nicorandil as an Adjunct to Reperfusion Therapy for Acute Myocardial Infarction : J-WIND Trials(Late Breaking Clinical Trials in Japan, The 71st Annual Scientific Meeting of the Japanese Circulation Soci
3 Human Atrial Natriuretic Peptide and Nicorandil as an Adjunct to Reperfusion Therapy for Acute Myocardial Infarction : Lessons from J-WIND Trials(Discovery of Adjunctive Therapies for Acute Myocardial Infarction-From Bench to Bedside-, The 71st Annual S
The ATP-sensitive Potassium Channel Opener, Nicorandil, Reduces No-reflow Phenomenon and Infarct Size in the Canine Heart
OJ-442 Gq-stimulation along with low activity of proteasome induces cardiac apoptosis : Potential mechanism of transition from hypertrophy to failure(Heart Failure, Basic 3 (M) : OJ54)(Oral Presentation (Japanese))
OJ-324 Antagonism of Aldosterone Receptor Attenuates Severity of Cardiac Dysfunction and Inflammation Induced by Chronic Blockade of NO Synthesis in Rats(Cardiovascular Pharmacology, Basic/Clinical 2 (H) : OJ39)(Oral Presentation (Japanese))
Canine DNA Array as a Potential Tool for Combining Physiology and Molecular Biology : An Application for the Gene Expression Profile of Regional Ischemic Myocardium
An Adenosine Analogue, Chloroadenosine, Attenautes Pressure-Overload Myocardial Hypertrophy in Mice
The New Metalloproteinase Inhibitor Blocks Cardiac Hypertrophy Induced by both Phenylephrine and Angiotensin II
OJ-020 Oxidative Stress-induced Decline of Proteasome Activity Promotes Accumulation of Proapoptotic Proteins and Contributes to Cardiomyocytes Apoptosis in Failing Heart(Heart failure, basic-2 (M) OJ4,Oral Presentation (Japanese),The 70th Anniversary Ann
The Pathophysiological Role of Endoplasmic Reticulum Stress in the Failing Hearts : Possibility of Brain-Type Natriuretic Peptide as its Marker (Heart Failure, Basic 6 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Decreased Proteasome Activity by Oxidative Stress Induces Cardiac Myocytes Apoptosis via p53-mediated Pathway(Heart Failure, Basic 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PE-541 Glycosaminoglycan Modification of Neuropilin-1 Modulates VEGFR2 Signaling(Molecular biology, vascular-3, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Differential Effect on Acute Cardioprotection against Myocardial Infarction by Statins : Role of ECTO-5'-Nucleotidase
Preischemic Protein Kinase-A Activation Triggers Cardioprotection of Ischemic Preconditioning via the Inhibition of Rho-Kinase
Rationale and Design of a Large-Scale Trial Using Nicorandil as an Adjunct to Percutaneous Coronary Intervention for ST-Segment Elevation Acute Myocardial Infarction : Japan-Working Groups of Acute Myocardial Infarction for the Reduction of Necrotic Damag
Rationale and Design of a Large-Scale Trial Using Atrial Natriuretic Peptide (ANP) as an Adjunct to Percutaneous Coronary Intervention for ST-Segment Elevation Acute Myocardial Infarction : Japan-Working Groups of Acute Myocardial Infarction for the Reduc
HB-EGF Is an Essential Growth Factor for Cardiomyocyte Metabolism : The Possibility for a Novel Therapeutic Target for Heart Failure
Rho-kinase Inhibitiors Administered After the Onset of Reperfusion Limit Infarct Size : A Novel Strategy for Adjunctive Therapies Against Myocardial Infarction
A Role of Transient Preischemic Activation of Protein Kinase-A in Ischemic Preconditioning in Vivo : Involvement of p38 Mitogen-activated Protein Kinase
Amlodipine Increases Coronary Blood Flow and Reduces the Severity of Myocardial Ischemia in Canine Hearts : The Role of NO and Adenosine
Inhibitory Effects of the CP Compound on Coronary Thromboemboli Caused by an Inhibition of Adenosine Receptors during Coronary Hypoperfusion
The Long-acting Calcium Channel Blockers Attenuate Cardiac Hypertrophy Induced by Chronic NO Inhibition : The Investigation of the Potential Subcellular Mechanisms
Gene Expression Profile of Failing Human Hearts In Patients with Dilated Cardiomyopathy Using "Gene Chip" Analysis

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