Shimazu Takashi | Department Of Internal Medicine And Therapeutics Osaka University Graduate School Of Medicine

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著作論文

Up-Regulation of Cell Adhesion Molecule Genes in Human Endothelial Cells Stimulated by Lymphotoxin Alpha : DNA Microarray Analysis
PJ-225 Lymphotoxin-alpha Regulates Monocyte Adhesion to Endothelial Cells by Up-regulation of Cell Adhesion Molecules via PI3K-Akt-NFkB Pathway in Human Endothelial Cells(Atherosclerosis, basic(06)(IHD),Poster Session(Japanese),The 72nd Annual Scientific
Inhibition of Cardiac Remodeling by Pravastatin Is Associated with Amelioration of Endoplasmic Reticulum Stress
Atorvastatin Slows the Progression of Cardiac Remodeling in Mice with Pressure Overload and Inhibits Epidermal Growth Factor Receptor Activation
OJ-250 A Novel Role of Histamine H2 Receptors in the Pathophysiology of Canine Heart Failure Induced by Sustained Rapid Pacing(Heart failure, basic-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-092 Chronic Stimulation of Adenosine A2b Receptors Begun after Myocardial Infarction Prevents Cardiac Remodeling in Rats(Heart Failure, Cardiac Remodeling and Cardiomyopathy 2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PJ-047 The Combined Therapy of Angiotensin Converting Enzyme Inhibitor, Angiotensin II Receptor Blocker and Beta-blockers Submaximally Limits Infarct Size(Acute Myocardial Infarction, Basic 2 (IHD) : PJ8)(Poster Session (Japanese))
PJ-046 Celiprolol, a Selective β_1-Adrenoceptor Antagonist, Limits Infarct Size via NO-dependent Mechanisms(Acute Myocardial Infarction, Basic 2 (IHD) : PJ8)(Poster Session (Japanese))
PJ-092 Activation of Adenosine A1 Receptors Inhibits Aldosterone Synthesis in Adrenal Glands(Metabolism/Biochemistry/Energetics-3 (M) PJ16,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
2 How to Find Novel Pathophysiology and Therapy of Chronic Heart Failure from Genomic Database(Perspectives of Genome-wide Study to Identify Genes Underlying Cardiovascular Diseases,Roundtable Discussion 10 (RT10) (M),The 70th Anniversary Annual Scientifi
OJ-442 Gq-stimulation along with low activity of proteasome induces cardiac apoptosis : Potential mechanism of transition from hypertrophy to failure(Heart Failure, Basic 3 (M) : OJ54)(Oral Presentation (Japanese))
OJ-324 Antagonism of Aldosterone Receptor Attenuates Severity of Cardiac Dysfunction and Inflammation Induced by Chronic Blockade of NO Synthesis in Rats(Cardiovascular Pharmacology, Basic/Clinical 2 (H) : OJ39)(Oral Presentation (Japanese))
Canine DNA Array as a Potential Tool for Combining Physiology and Molecular Biology : An Application for the Gene Expression Profile of Regional Ischemic Myocardium
OJ-320 Granulocyte Colony-Stimulating Factor Mediates Cardioprotection against Ischemia/Reperfusion Injury via Phosphatidylinositol-3-Kinase/Akt Pathway : Evaluation in Vitro Cardiomyocytes and in Vivo Canine Hearts(Acute myocardial infarction, basic-2 (I
OJ-020 Oxidative Stress-induced Decline of Proteasome Activity Promotes Accumulation of Proapoptotic Proteins and Contributes to Cardiomyocytes Apoptosis in Failing Heart(Heart failure, basic-2 (M) OJ4,Oral Presentation (Japanese),The 70th Anniversary Ann
The Pathophysiological Role of Endoplasmic Reticulum Stress in the Failing Hearts : Possibility of Brain-Type Natriuretic Peptide as its Marker (Heart Failure, Basic 6 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Selective Blockade of Serotonin 5HT2A Receptor Increases Coronary Blood Flow via NO-Dependent Pathway Through 5HT1B Receptor in Hypoperfused Canine Hearts(Coronary Circulation, Basic/Clinical 3 (IHD), The 69th Annual Scientific Meeting of the Japanese Cir
Decreased Proteasome Activity by Oxidative Stress Induces Cardiac Myocytes Apoptosis via p53-mediated Pathway(Heart Failure, Basic 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PE-541 Glycosaminoglycan Modification of Neuropilin-1 Modulates VEGFR2 Signaling(Molecular biology, vascular-3, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-126 The Direct Physiological Impact of C-reactive Protein on Ischemicin Vivo Hearts : Reduced Cardiac Nitric Oxide Production but Saved Metabolization(Coronary Circulation, Basic/Clinical 2 (IHD) : OE15)(Oral Presentation (English))

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