Mitochondrial Respiration of Experimentally Produced Ischemic Heart Muscle in Dogs

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概要

• 論文の詳細を見る

• The mitochondrial respiration in experimentally produced myocardial infarction or ischemic heart was investigated. In myocardial infarction, disturbances of respiration appeared 15min. after the coronary artery occlusion, which was recognized as the deterioration of respiratory control ratio as result of the acceleration of oxygen consumption rate in state 4. Deterioration of respiratory control ratio was also recognized in mitochondria of the heart which was stimulated electrically, but was not recognized in the heart mitochondria which were prepared from the heart stored in anoxic state without beating.Magnesium-induced ATPase activity of the infarcted heart mitochondria was higher than that of the intact heart mitochondria.It was assumed from these results that disturbance of the respiration of infarcted heart mitochondria was attributed to the uncoupling of oxidative phosphorylation.

• International Heart Journal刊行会の論文

著者

• Yamamura Yuichi

  Third Department of Internal Medicine, Osaka University Medical School

• Yamagami Toru

  The Center For Adult Diseases.

• Yamagami Toru

  Third Department Of Internal Medicine Medical School Of Osaka University

• MORITA Yoshiharu

  Third Dept. of Intern. Med., Osaka Univ. Med. School

• YAMAMURA Yuichi

  Third Department of Internal Medicine, Medical School of Osaka University

• YAMAMURA YUICHI

  Third Department of Internal Medicine and Central Laboratory, Medical School of Osaka University

• YAMAGAMI Toru

  Third Department of Internal Medicine, Medical School of Osaka University

• MORITA Yoshiharu

  Third Department of Internal Medicine, Medical School of Osaka University

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