ビワがんしゅ病の樹冠内および樹園内における拡散

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A loquat orchard with an area of 150 m^2 planted with sixty-three trees (7×9) of one-year-old healthy loquat in 1975 was used for this experiment during twelve years. First, one loquat plant laid in the center of the orchard was inoculated with a streptomycin-tolerant strain of Pseudomonas syringae pv. eriobotryae, the causal bacterium of loquat canker. Thereafter, the spread of the canker disease from the inoculated plant to other plants in the orchard and the seasonal spread of the pathogen into buds, leaves, twigs and other parts of the infected plants were traced under natural condition without controling by pesticides or pruning by secateurs during thirteen years from 1975 to 1987. From one lesion on the loquat inoculated in 1975, 548 lesions developed, and total numbers of lesions on whole parts of all infected loquat trees were 5,268 in 1987. Especially, lesion numbers increased rapidly during first four years after planting. More than eighty percent of these lesions originated from sprouts in spring and wounded parts injured by harvesting or disbudding. About seventy percent of initial infection lesions was first appeared at sprouts in spring. Initial lesions on young trees, prior to the beginning of harvesting and disbudding developed into typical canker lesions on trunks and primary scaffold branches. Initial lesions on adult trees after the stage of the beginning of harvesting and disbudding also developed from wounded tissue by these works and frequently progressed to lateral branches.
日本植物病理学会の論文
1991-07-25

ビワがんしゅ病の樹冠内および樹園内における拡散

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