The role of cytochrome c and mitochondria in the signal transduction pathway for radiation-induced apoptosis
スポンサーリンク
概要
著者
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Matsubara Sho
Particle Radiation Oncology Research Center Research Reactor Institute Kyoto University
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Matsuura Shinya
京都大学放射線生物研究センター
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MATSUBARA Sho
Yokohama City Univ. School of MeD. Dep. of Radiol. Tokyo Med. &Dent. Univ. School of Dent. Dep. of R
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NAKAGAMI Yoshihiro
Yokohama city Univ.
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KURIHARA Hiroaki
Yokohama city Univ.
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TORIGOE Soichiro
Yokohama city Univ.
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Matsubara Sho
Yokohama City Univ. Dept. Radiol.
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Kurihara Hiroaki
Yokohama City Univ. Dept. Radiol.
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Torigoe Soichiro
Yokohama City Univ. Dept. Radiol.
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Matsubara Sho
Yokohama City Univ. School Of Med. Dep. Of Radiol. Tokyo Med. &dent. Univ. School Of Dent. Dep.
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Nakagami Yoshihiro
Yokohama City Univ. Dept. Radiol.
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Matsubara Sho
Dept. Of Radiology School Of Medicine Yokohama City Univ.
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Matsushita Satoru
Department Of Technical Support And Development Natl. Inst. Radiol. Sci.
関連論文
- 242 Analysis of apoptosis induction in Nbs1 deficient cells(Apoptosis related, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- 206 Functional Domain Analysis of NBS1 Gene in Homologous Recombinational Repair(Physics, chemistry and DNA damage, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- 98 Localization of Rad51 protein in cells from patients with Fanconi Anemia D1 group(Repair of radiation damage, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- S-3-1 Mechanisms of DNA double-strand break repair(Biological Effects of Low Dose and Low Dose-rate Irradiations, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- Mutation screening for a novel cancer-predisposition syndrome of total PCS
- p53-Binding Protein 1 (53BP1) is Involved in the Repair of DNA Double-Strand Breaks
- Analysis of the function of NBS1 in DNA double-strand break repair
- Histone H2AX regulates the formation of NBS1 foci on DNA damage sites
- Dissociation of ATM/NBS1 functions in telomere maintenance
- NBS1 is involved in recognition of DNA damage and initiation of homologous recombination.
- Bystander mutagenic effect of alpha particle in human-hamster hybrid cells
- Somatic Mutation Caused by Low Dose Rate Tritium Radiation : Studies Using A Hyper-sensitive DetectionSystem
- Complementation studies of immortalized cell lines from infants with total PCS
- Establishement of mouse Nbs1-deficient cell lines by gene targeting
- Association of cells from Fanconi anemia patients with radiation sensitivity
- Association of DNA repair protein NBS1 with telomere maintenance
- The function of NBS1 and histone H2AX in the early process of DNA repair.
- Reversed dose-rate effect of high LET radiation in mutation induction
- Identification of a novel mRNA containing GADD45 exons
- Molecular Genetic Diagnosis of von Hippel-Lindau Disease: Analysis of Five Japanese Families
- X-Ray Dose Enhancement Caused by the Addition of Gd-contrast Media and its Effect for Chromosome Aberration Efficiencies.
- The Relationship Between Residual Interphase Chromosome Breakage and Cellular Radiosensitivity in Transformed Syrian Golden Hamster Cells
- A Comparison of Biological Responses of Human Tumor Cells After Irradiation with Heavy Ions and Neutrons
- Response of human tumor cells in plateau phase and grown as spheroids to carbon beam irradiation.
- Four novel mutations of the Fanconi anemia group A gene (FAA) in Japanese patients
- Functionally important domain analysis of the Nijimegen breakage syndrome gene. NBS1
- Development of a Radio-sensitive Mutation System for the Study of Dose Rate Effects of Space High LET Radiation
- Responses of total and quiescent cell populations in solid tumors to carbon ion beam irradiation (290 MeV/u) in vivo
- The role of cytochrome c and mitochondria in the signal transduction pathway for radiation-induced apoptosis
- GADD45 and JNK/SAPK expressions following treatment of high osmolarity agent and γray in HeLa S3 cells
- Evaluation of Late Radiation Damage of Normal Tissues in Mouse Hind Legs after Irradiation with Carbon Beams
- RBE and Dose Distribution of Heavy Ion Beams.
- Cytosol extracted from irradiated cells induced DNA fragmentation
- Time Dependent Reduction of Skin Shrinkage in Mouse after Irradiation with the Falloff of Carbon Bragg Peak
- Dose response relations in acute cell death of pancreatic islets after irradiation of golden hamster with X-ray ,7OMeV proton and 290MeV carbon beams.
- Morphological Evaluation of Normal Tissue Damage after Neon Beam Irradiation in Mouse Hind Legs
- Evaluation of Normal Tissue Damage after Fractionated Irradiation of HIMAC Carbon- 12 in Mouse Hind Legs
- RBBs of 30MeV Fast Neutrons and 135MeV Carbon Beams for Clongenecities in Mouse Growing Tail Cartilage Cells.
- The effects of cell death and mutatlon induction lrradiated with acceterated carbon ion beams with spread out Bragg peak.
- Nijmegen Breakage Syndrome (NBS) gene, NBS1, is involved in telomeric length maintenance.
- Skin reaction after single doses of HIMAC Carbon-12 Spread- Out-Bragg-Peak
- Mapping of the underlying gene for rare genetic complementation group Fanconi anemia
- NBS1 Regulates Apoptosis Induced by γ-Irradiation in Lymphoblastoid
- Mutation screening of the NBS1 gene in sporadic malignant lymphoma
- Morphometric Evaluation of Late Normal Tissue Damage after Neon Beam Irradiation in Mouse Hind Legs
- RBE of Heavy Ions (proton, carbon,neon) for Acute Cell Death of Pancreatic Islet Cells of the Golden Hamster(Mesocricetus auratus)
- O-15. Chromosome instability and cancer predisposition in the PCS (premature chromatid separation) syndrome(Abstracts of the oral and poster presentations,Abstracts of the 55th Annual Meeting of the Society of Chromosome Research)
- O-11. The optimal conditions for cytological diagnosis of total premature chromatid separation (PCS), a novel cancer-prone genetic trait associated with mitotic checkpoint defect(Abstracts of the oral and poster presentations)(The 53rd Annual Meeting of t
- Morphological Evaiuation of Normal Tissue Damage after Carbon Beam Irradiation in Mouse Hind Legs
- A polymorphic CA repeat marker at the human 27-kD calbindin (CALB1) locus