SAP increases FynT kinase activity and is required for phosphorylation of SLAM and Ly9
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概要
- 論文の詳細を見る
- 2004-05-01
著者
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HOWIE Duncan
Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School
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TERHORST Cox
Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School
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Terhorst Cox
Division Of Immunology Beth Israel Deaconess Medical Center
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Poy Florence
Department Of Cancer Biology Dana Farber Cancer Institute Harvard Medical School
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Lanyi Arpad
Division Of Immunology Beth Israel Deaconess Medical Center Harvard Medical School
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Howie Duncan
Division Of Immunology Beth Israel Deaconess Medical Center Harvard Medical School
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SIMARRO Maria
Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School
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BRUGGEMAN Joost
Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School
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CHOI Michelle
Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School
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SUMEGI Janos
Division of Hematology and Oncology, Cincinnati Children's Hospital Medical Center
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ECK Michael
Department of Cancer Biology, Dana Farber Cancer Institute, Harvard Medical School
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Eck Michael
Department Of Cancer Biology Dana Farber Cancer Institute Harvard Medical School
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Sumegi Janos
Division Of Hematology And Oncology Cincinnati Children's Hospital Medical Center
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Choi Michelle
Division Of Immunology Beth Israel Deaconess Medical Center Harvard Medical School
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Simarro Maria
Division Of Immunology Beth Israel Deaconess Medical Center Harvard Medical School
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Bruggeman Joost
Division Of Immunology Beth Israel Deaconess Medical Center Harvard Medical School
関連論文
- Potential pathways for regulation of NK and T cell responses: differential X-linked lymphoproliferative syndrome gene product SAP interactions with SLAM and 2B4
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- Generation of antigen-specific cytotoxic T lymphocytes and regulation of cytokine production takes place in the absence of CD3ζ
- Expression of a CD3ε transgene in CD3εnull mice does not restore CD3γ and δ expression bur efficienly rescues T cell development from a subpopulation of prothymocytes
- Over-expression of CD3ε transgenes blocks T lymphocyte development
- Selection of peripheral and intestinal T lymphocytes lacking CD3 ζ
- Natural killer cell development is bloked in the context of aberrant T lymphocyte ontogeny
- SAP increases FynT kinase activity and is required for phosphorylation of SLAM and Ly9
- Role of the CD5 molecule on TCR γδ T cell-mediated immune functions: development of germinal centers and chronic intestinal inflammation
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