TERHORST Cox | Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School

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著作論文

Potential pathways for regulation of NK and T cell responses: differential X-linked lymphoproliferative syndrome gene product SAP interactions with SLAM and 2B4
Activation-induced apoptosis of mature T cells is dependent upon the level of surface TCR but not on the presende of the CD3ζ ITAM
Generation of antigen-specific cytotoxic T lymphocytes and regulation of cytokine production takes place in the absence of CD3ζ
Expression of a CD3ε transgene in CD3εnull mice does not restore CD3γ and δ expression bur efficienly rescues T cell development from a subpopulation of prothymocytes
Over-expression of CD3ε transgenes blocks T lymphocyte development
Selection of peripheral and intestinal T lymphocytes lacking CD3 ζ
Natural killer cell development is bloked in the context of aberrant T lymphocyte ontogeny
SAP increases FynT kinase activity and is required for phosphorylation of SLAM and Ly9
Composition of TCR-CD3 complex in human intestinal intraepithelial lymphocytes: lack of Fc_εRI_γ chain
The interchain disulfide bond between TCRαβ heterodimers on human T cells is not required for TCR-CD3 membrane expression and signal transduction
GITR engagement preferentially enhances proliferation of functionally competent CD4^+CD25^+FoxP3^+ regulatory T cells
Blocking inducible co-stimulator in the absence of CD28 impairs T_h1 and CD25^+ regulatory T cells in murine colitis

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