Potential pathways for regulation of NK and T cell responses: differential X-linked lymphoproliferative syndrome gene product SAP interactions with SLAM and 2B4
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概要
- 論文の詳細を見る
- 2001-12-01
著者
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SAYOS Joan
Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School
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NGUYEN B.
Department of Molecular Microbiology and Immunology, Brown University
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WU Chengbin
Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School
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STEPP E.
Department of Pathology, University of Texas Southwestern Medical Center
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HOWIE Duncan
Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School
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SCHATZLE D.
Department of Pathology, University of Texas Southwestern Medical Center
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KUMAR Vinay
Department of Pathology, University of Texas Southwestern Medical Center
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BIRON A.
Department of Molecular Microbiology and Immunology, Brown University
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TERHORST Cox
Division of Immunology, Beth Israel Deaconess Medical Center, Harvard Medical School
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Terhorst Cox
Division Of Immunology Beth Israel Deaconess Medical Center Harvard Medical School
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Terhorst Cox
Division Of Immunology Beth Israel Deaconess Medical Center
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Biron A.
Department Of Molecular Microbiology And Immunology Brown University
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Stepp E.
Department Of Pathology University Of Texas Southwestern Medical Center
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Sayos Joan
Division Of Immunology Beth Israel Deaconess Medical Center Harvard Medical School
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Wu Chengbin
Division Of Immunology Beth Israel Deaconess Medical Center Harvard Medical School
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Schatzle D.
Department Of Pathology University Of Texas Southwestern Medical Center
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Kumar V
La Jolla Inst. Allergy And Immunology Ca Usa
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Howie D
Harvard Medical School Ma Usa
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Howie Duncan
Division Of Immunology Beth Israel Deaconess Medical Center Harvard Medical School
関連論文
- Potential pathways for regulation of NK and T cell responses: differential X-linked lymphoproliferative syndrome gene product SAP interactions with SLAM and 2B4
- Activation-induced apoptosis of mature T cells is dependent upon the level of surface TCR but not on the presende of the CD3ζ ITAM
- Generation of antigen-specific cytotoxic T lymphocytes and regulation of cytokine production takes place in the absence of CD3ζ
- Expression of a CD3ε transgene in CD3εnull mice does not restore CD3γ and δ expression bur efficienly rescues T cell development from a subpopulation of prothymocytes
- Over-expression of CD3ε transgenes blocks T lymphocyte development
- Selection of peripheral and intestinal T lymphocytes lacking CD3 ζ
- Natural killer cell development is bloked in the context of aberrant T lymphocyte ontogeny
- SAP increases FynT kinase activity and is required for phosphorylation of SLAM and Ly9
- Role of the CD5 molecule on TCR γδ T cell-mediated immune functions: development of germinal centers and chronic intestinal inflammation
- Composition of TCR-CD3 complex in human intestinal intraepithelial lymphocytes: lack of Fc_εRI_γ chain
- The interchain disulfide bond between TCRαβ heterodimers on human T cells is not required for TCR-CD3 membrane expression and signal transduction
- GITR engagement preferentially enhances proliferation of functionally competent CD4^+CD25^+FoxP3^+ regulatory T cells
- Blocking inducible co-stimulator in the absence of CD28 impairs T_h1 and CD25^+ regulatory T cells in murine colitis