Regulation of the E_β gene in vivo: lessons from E_β^d transgenic mice
スポンサーリンク
概要
著者
-
Glimcher Laurie
Department Of Immunology And Infections Diseases Harvard School Of Public Health Harvard Medical Sch
-
Glimcher Laurie
Department Of Cancer Biology Harvard School Of Public Health
-
DOUHAN III
Department of Cancer Biology, Harvard School of Public Health
-
BROWN Julia
Department of Cancer Biology, Harvard School of Public Health
-
GLEIT Zachary
Department of Cancer Biology, Harvard School of Public Health
-
DAVID Chella
Department of Immunology, Mayo Clinic and Mayo Graduate School of Medicine
-
Douhan Iii
Department Of Cancer Biology Harvard School Of Public Health
-
Brown Julia
Department Of Cancer Biology Harvard School Of Public Health
-
David Chella
Department Of Immunology Mayo Clinic
-
David Chella
Department Of Immunology Mayo Clinic And Mayo Graduate School Of Medicine
-
Gleit Zachary
Department Of Cancer Biology Harvard School Of Public Health
関連論文
- Marking IL-4-producing cells by knock-in of the IL-4 gene
- Regulation of the E_β gene in vivo: lessons from E_β^d transgenic mice
- B7-2 (CD86) is essential for the development of IL-4-producing T cells
- A minimal level of MHC class II expression is sufficient to abtogate autoreactivity
- Transgenic mice expressing MHC class II molecules with truncated A_β cytoplasmic domains reveal signaling-independent defects in antigen presentation
- Asthmatic changes in mice lacking T-bet are mediated by IL-13
- Trypanosoma cruzi infection in MHC-deficient mice:further evidence for the role of both class I-and class II-restricted T cells in immune resistance and disease
- Decreased immediate inflammatory gene induction in activating transcription factor-2 mutant mice
- MHC class II-independent and -dependent T cell expansion and B cell hyperactivity in vivo in mice deficient in CD152 (CTLA-4)
- In vivo staphylococcal superantigen-driven polyclonal lg responses in mice : dependence upon CD4^+ cells and human MHC class II
- V_β T cells protect from demyelinating disease in a viral model of multiple sclerosis
- Expression of an ovalbumin-soecific V_β8.2 TCR transgene inhibits collagen arthritis in B10.Q mice
- Autoimmune diabetes in HLA-DR3/DQ8 transgenic mice expressing the co-stimulatory molecule B7-1 in the β cells of islets of Langerhans
- HLA class II transgenic mice authenticate restriction of myelin oligodendrocyte glycoprotein-specific immune response implicated in multiple sclerosis pathogenesis
- Expression of the H2-E molecule mediates protection to collagen-induced arthritis in HLA-DQ8 transgenic mice: role of cytokines
- Modulation of HLA-DQ-restricted collagen-induced arthritis by HLA-DRB1 polymorphism
- Staphylococcal enterotoxin B-induced activation and concomitant resistance to cell death in CD28-deficient HLA-DQ8 transgenic mice