Trypanosoma cruzi infection in MHC-deficient mice:further evidence for the role of both class I-and class II-restricted T cells in immune resistance and disease
スポンサーリンク
概要
著者
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Glimcher Laurie
Department Of Immunology And Infections Diseases Harvard School Of Public Health Harvard Medical Sch
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Glimcher Laurie
Department Of Cancer Biology Harvard School Of Public Health
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GRUSBY Michael
Department of lmmunology and Infectious Diseases, Harvrd School of Public Health
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TARLETON Rick
Department of Cellular Biology, University of Georgia
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POSTAN Miriam
Department of Cellular Biology, University of Georgia
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Postan Miriam
Department Of Cellular Biology University Of Georgia
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Tarleton Rick
Department Of Cellular Biology University Of Georgia
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Grusby Michael
Department Of Cancer Biology Harvard School Of Public Health
関連論文
- Marking IL-4-producing cells by knock-in of the IL-4 gene
- Regulation of the E_β gene in vivo: lessons from E_β^d transgenic mice
- B7-2 (CD86) is essential for the development of IL-4-producing T cells
- A minimal level of MHC class II expression is sufficient to abtogate autoreactivity
- Transgenic mice expressing MHC class II molecules with truncated A_β cytoplasmic domains reveal signaling-independent defects in antigen presentation
- Asthmatic changes in mice lacking T-bet are mediated by IL-13
- Trypanosoma cruzi infection in MHC-deficient mice:further evidence for the role of both class I-and class II-restricted T cells in immune resistance and disease
- Decreased immediate inflammatory gene induction in activating transcription factor-2 mutant mice