Marking IL-4-producing cells by knock-in of the IL-4 gene
スポンサーリンク
概要
著者
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Glimcher L
Harvard Medical School Ma Usa
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Glimcher L
Department Of Lmmunology And Infectious Diseases Harvrd School Of Public Health
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Glimcher Laurie
Department Of Immunology And Infections Diseases Harvard School Of Public Health Harvard Medical Sch
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Glimcher Laurie
Department Of Cancer Biology Harvard School Of Public Health
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HO I-Cheng
Department of lmmunology and Infectious Diseases, Harvrd School of Public Health
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KAPLAN Mark
Department of lmmunology and Infectious Diseases, Harvrd School of Public Health
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JACKSON-GRUSBY Laurie
The Whitehead Institute for Biomedical Research
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GRUSBY Michael
Department of lmmunology and Infectious Diseases, Harvrd School of Public Health
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Ho I-cheng
Department Of Lmmunology And Infectious Diseases Harvrd School Of Public Health
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Kaplan Mark
Department Of Lmmunology And Infectious Diseases Harvrd School Of Public Health
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Grusby M
Department Of Lmmunology And Infectious Diseases Harvrd School Of Public Health
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Grusby Michael
Department Of Cancer Biology Harvard School Of Public Health
関連論文
- Marking IL-4-producing cells by knock-in of the IL-4 gene
- Regulation of the E_β gene in vivo: lessons from E_β^d transgenic mice
- B7-2 (CD86) is essential for the development of IL-4-producing T cells
- A minimal level of MHC class II expression is sufficient to abtogate autoreactivity
- Transgenic mice expressing MHC class II molecules with truncated A_β cytoplasmic domains reveal signaling-independent defects in antigen presentation
- Asthmatic changes in mice lacking T-bet are mediated by IL-13
- Trypanosoma cruzi infection in MHC-deficient mice:further evidence for the role of both class I-and class II-restricted T cells in immune resistance and disease
- Decreased immediate inflammatory gene induction in activating transcription factor-2 mutant mice