Glimcher L | Harvard Medical School Ma Usa
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概要
関連著者
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Glimcher L
Harvard Medical School Ma Usa
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Glimcher Laurie
Department Of Immunology And Infections Diseases Harvard School Of Public Health Harvard Medical Sch
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Glimcher Laurie
Department Of Cancer Biology Harvard School Of Public Health
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GRUSBY Michael
Department of lmmunology and Infectious Diseases, Harvrd School of Public Health
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LO David
Department of Immunology, The Scripps Research Institute
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LAUFER Terri
Department of Immunology and Infectious Diseases, Harvard School of Public Health
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Lo David
Department Of Immunology The Scripps Research Institute
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Laufer Terri
Department Of Immunology And Infectious Diseases Harvard School Of Public Health
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Grusby Michael
Department Of Cancer Biology Harvard School Of Public Health
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Glimcher L
Department Of Lmmunology And Infectious Diseases Harvrd School Of Public Health
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HO I-Cheng
Department of lmmunology and Infectious Diseases, Harvrd School of Public Health
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KAPLAN Mark
Department of lmmunology and Infectious Diseases, Harvrd School of Public Health
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JACKSON-GRUSBY Laurie
The Whitehead Institute for Biomedical Research
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de BONT
Department of Immunology and Infectious Diseases, Harvard School of Public Health
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REILLY Christina
Department of Immunology, The Scripps Research Institute
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SMILEY Stephen
Department of Cancer Biology, Harvard School of Public Health
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De Bont
Department Of Immunology And Infectious Diseases Harvard School Of Public Health
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Ho I-cheng
Department Of Lmmunology And Infectious Diseases Harvrd School Of Public Health
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Kaplan Mark
Department Of Lmmunology And Infectious Diseases Harvrd School Of Public Health
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Smiley Stephen
Department Of Cancer Biology Harvard School Of Public Health
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Grusby M
Department Of Lmmunology And Infectious Diseases Harvrd School Of Public Health
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Reilly Christina
Department Of Immunology The Scripps Research Institute
著作論文
- Marking IL-4-producing cells by knock-in of the IL-4 gene
- A minimal level of MHC class II expression is sufficient to abtogate autoreactivity
- Transgenic mice expressing MHC class II molecules with truncated A_β cytoplasmic domains reveal signaling-independent defects in antigen presentation