Expression of the H2-E molecule mediates protection to collagen-induced arthritis in HLA-DQ8 transgenic mice: role of cytokines
スポンサーリンク
概要
著者
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David Chella
Department Of Immunology Mayo Clinic
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Harvinder Luthra
Department Of Rheumatology Mayo Clinic
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Griffiths Marie
Ressearch Service Veterans Administration Medical Center And Department Fo Medicine University Of Ut
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TANEJA Veana
Department of Immunology Mayo Clinic
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HANSEN Julie
Department of Immunology Mayo Clinic
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SMART Michele
Department of Immunology Mayo Clinic
関連論文
- Regulation of the E_β gene in vivo: lessons from E_β^d transgenic mice
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- V_β T cells protect from demyelinating disease in a viral model of multiple sclerosis
- Expression of an ovalbumin-soecific V_β8.2 TCR transgene inhibits collagen arthritis in B10.Q mice
- Autoimmune diabetes in HLA-DR3/DQ8 transgenic mice expressing the co-stimulatory molecule B7-1 in the β cells of islets of Langerhans
- HLA class II transgenic mice authenticate restriction of myelin oligodendrocyte glycoprotein-specific immune response implicated in multiple sclerosis pathogenesis
- Expression of the H2-E molecule mediates protection to collagen-induced arthritis in HLA-DQ8 transgenic mice: role of cytokines
- Modulation of HLA-DQ-restricted collagen-induced arthritis by HLA-DRB1 polymorphism
- Staphylococcal enterotoxin B-induced activation and concomitant resistance to cell death in CD28-deficient HLA-DQ8 transgenic mice