B cell-mediated down-regulation of IFN-γ and IL-12 Production induced during antitumor immune responses in the tumorbearing state
スポンサーリンク
概要
著者
-
HAMAOKA TOSHIYUKI
Biomedical Research Center, Faculty of Medicine, Osaka University
-
HAMAOKA Toshiyuki
Department of Oncology (C6), Osaka University Graduate School of Medicine
-
Ono Shiro
Division Of Oncogenesis Department Of Oncology Biomedical Research Center Osaka University Graduate
-
Fujiwara H
Department Of Oncology Osaka University Graduate School Of Medicine
-
Fujiwara Hiromi
Department Of Oncology (c6) Graduate School Of Medicine Osaka University
-
MARUO Seiji
Biomedical Research Center, Osaka University Medical School
-
FUJIWARA Hiromi
Biomedical Research Center, Osaka University Medical School
-
Fujiwara H
Osaka Univ. Osaka Jpn
-
Maruo Seiji
Biomedical Research Center Osaka University Medical School
-
WIJESURIYA Rishani
Department of Oncology, Biomedical Research Center, Osaka University Graduate School of Medicine
-
ZOU Jian-Ping
Biomedical Research Center, Osaka University Medical School
-
OGAWA Makoto
Biomedical Research Center, Osaka University Medical School
-
UMEHARA Kazunari
Biomedical Research Center, Osaka University Medical School
-
WIJESURIYA Rishani
Biomedical Research Center, Osaka University Medical School
-
ONO Shiro
Biomedical Research Center, Osaka University Medical School
-
YAMASHITA Motozo
Biomedical Research Center, Osaka University Medical School
-
Ono Shiro
Department Of Oncology (c6) Graduate School Of Medicine Osaka University
-
Yamashita Motozo
Biomedical Research Center Osaka University Medical School
-
Hamaoka Toshiyuki
Department Of Oncology (c6) Osaka University Graduate School Of Medicine
-
Hamaoka Toshiyuki
Biomedical Research Center Faculty Of Medicine Osaka University
-
Hamaoka Toshiyuki
Biomedical Research Center Osaka University Medical School
-
Zou Jian-ping
Biomedical Research Center Osaka University Medical School
-
Fujiwara Hiromi
Biomedical Research Center Osaka University Medical School
-
Fujiwara H
Department Of Oncology (c6) Graduate School Of Medicine Osaka University
-
Fujiwara Hiromi
Department Of Oncology Osaka University Graduate School Of Medicine
-
Hamaoka Toshiyuki
Department Of Oncology Osaka University Graduate School Of Medicine
-
Ogawa Makoto
Biomedical Research Center Osaka University Medical School
-
Fujiwara H
Department Of Oncology Biomedical Research Center Osaka University Graduate School Of Medicine
-
Wijesuriya Rishani
Department Of Oncology Biomedical Research Center Osaka University Graduate School Of Medicine
-
Umehara Kazunari
Biomedical Research Center Osaka University Medical School
関連論文
- SCID (Severe Combined Immunodeficiency) Mice as a New System to Investigate Metastasis of Human Tumors
- A mechanism underlying STAT4-mediated up-regulation of IFN-γ induction in TCR-triggered T cells
- Reversible CD8 expression induced by common cytokine receptor γchain-dependent cytokines in a cloned CD4^+ T_h1 cell line
- Requirement of IFN-γ in IL-12 production induced by collaboration between V_α14^+ NKT cells and antigen-presenting cells
- OJ-078 Impact of Gender Differences in Prevalence of Risk Factors in Patients with Atrial Fibrillation in Japan : Observations from J-TRACE Study(OJ13,Arrhythmia, Others (Clinical/Diagnosis/Treatment) 1 (A),Oral Presentation (Japanese),The 73rd Annual Sci
- CD28 co-stimulatory signals induce IL-2 receptor expression on antigen-stimulated virgin T cells by an IL-2-independent mechanism
- B7.2-Ig fusion proteins enhance IL-4-dependent differentiation of tumor-sensitized CD8^+ cytotoxic T lymphocyte precursors
- The capacity of the natural ligands for CD28 to drive IL-4 expression in naive and antigen-primed CD4^+ and CD8^+ T cells
- Histiocytoid breast carcinoma : Solid variant of invasive lobular carcinoma with decreased expression of both E-cadherin and CD44 epithelial variant
- Differential IL-12 responsiveness of T cells but not of NK cells from tumor-bearing mice in IL-12-respomsive versus-unresponsive tumor models
- MOLECULAR MECHANISMS UNDERLYING T CELL MIGRATION RESPONSIBLE FOR IL-12-INDUCED TUMOR REGRESSION
- Molecular mechanisms underlying IFN-γ-mediated tumor growth inhibition induced during tumor immunotherapy with rIL-12
- B cell-mediated down-regulation of IFN-γ and IL-12 Production induced during antitumor immune responses in the tumorbearing state
- The involvement of neutrophils in the resistance to Leishmania major infection in susceptible but not in resistant mice
- Association of a tetraspanin CD9 with CD5 on the T cell surface: role of particular transmembrane domains in the association
- Inhibition of Growth and Metastasis of Ovarian Carcinoma by Administering a Drug Capable of Interfering with Vascular Endothelial Growth Factor Activity
- Opposite Actions of Transforming growth Factor-β_1 on the Gene Expression of Atrial Natriuretic Peptide Biological and Clearance Receptors in a Murine Thymic Stromal Cell Line
- A Defect in Cell-to-cell Adhesion via Integrin-Fibronectin Interactions in a Highly Metastatic Tumor Cell Line
- Do Structural Changes of T Cell Receptor Complex Occur in Tumor-bearing State?
- Systemic administration of rIL-12 induces complete tumor regression and protective immunity: response is correlated with a striking reversal of suppressed IFN-γ production by anti-tumor T cells
- Translocation of tyrosine-phosphorylated TCRζ chain to glycolipid-enriched membrane domains upons T cell activation
- Cyclosporin A inhibits late steps of T lymphocyte activation after transmembrane signaling.