Fujiwara H | Department Of Oncology (c6) Graduate School Of Medicine Osaka University

スポンサーリンク

概要

関連著者

もっと見る

著作論文

A mechanism underlying STAT4-mediated up-regulation of IFN-γ induction in TCR-triggered T cells
Reversible CD8 expression induced by common cytokine receptor γchain-dependent cytokines in a cloned CD4^+ T_h1 cell line
Requirement of IFN-γ in IL-12 production induced by collaboration between V_α14^+ NKT cells and antigen-presenting cells
CD28 co-stimulatory signals induce IL-2 receptor expression on antigen-stimulated virgin T cells by an IL-2-independent mechanism
B7.2-Ig fusion proteins enhance IL-4-dependent differentiation of tumor-sensitized CD8^+ cytotoxic T lymphocyte precursors
The capacity of the natural ligands for CD28 to drive IL-4 expression in naive and antigen-primed CD4^+ and CD8^+ T cells
Differential IL-12 responsiveness of T cells but not of NK cells from tumor-bearing mice in IL-12-respomsive versus-unresponsive tumor models
MOLECULAR MECHANISMS UNDERLYING T CELL MIGRATION RESPONSIBLE FOR IL-12-INDUCED TUMOR REGRESSION
Molecular mechanisms underlying IFN-γ-mediated tumor growth inhibition induced during tumor immunotherapy with rIL-12
B cell-mediated down-regulation of IFN-γ and IL-12 Production induced during antitumor immune responses in the tumorbearing state
Association of a tetraspanin CD9 with CD5 on the T cell surface: role of particular transmembrane domains in the association
Inhibition of Growth and Metastasis of Ovarian Carcinoma by Administering a Drug Capable of Interfering with Vascular Endothelial Growth Factor Activity
A Defect in Cell-to-cell Adhesion via Integrin-Fibronectin Interactions in a Highly Metastatic Tumor Cell Line
Do Structural Changes of T Cell Receptor Complex Occur in Tumor-bearing State?
Systemic administration of rIL-12 induces complete tumor regression and protective immunity: response is correlated with a striking reversal of suppressed IFN-γ production by anti-tumor T cells

もっと見る

スポンサーリンク