Molecular mechanisms underlying IFN-γ-mediated tumor growth inhibition induced during tumor immunotherapy with rIL-12
スポンサーリンク
概要
著者
-
HAMAOKA TOSHIYUKI
Biomedical Research Center, Faculty of Medicine, Osaka University
-
HAMAOKA Toshiyuki
Department of Oncology (C6), Osaka University Graduate School of Medicine
-
TAI Xu-Guang
Department of Oncology, Osaka University Graduate School of Medicine
-
Tai Xu-guang
Department Of Oncology Osaka University Graduate School Of Medicine
-
Fujiwara H
Department Of Oncology Osaka University Graduate School Of Medicine
-
Fujiwara Hiromi
Department Of Oncology (c6) Graduate School Of Medicine Osaka University
-
YAMAMOTO Norihiko
Biomedical Research Center, Osaka University Medical School
-
TAI Xu-Guang
Biomedical Research Center, Osaka University Medical School
-
FUJIWARA Hiromi
Biomedical Research Center, Osaka University Medical School
-
Fujiwara H
Osaka Univ. Osaka Jpn
-
Yu Wen-gong
Department Of Oncology Biomedical Research Center Osaka University Graduate School Of Medicine
-
WIJESURIYA Rishani
Department of Oncology, Biomedical Research Center, Osaka University Graduate School of Medicine
-
ZOU Jian-Ping
Biomedical Research Center, Osaka University Medical School
-
OGAWA Makoto
Biomedical Research Center, Osaka University Medical School
-
YU Wen-Gong
Biomedical Research Center, Osaka University Medical School
-
WIJESURIYA Rishani
Biomedical Research Center, Osaka University Medical School
-
TAKENAKA Hiroshi
Biomedical Research Center, Osaka University, Medical School
-
MU Jie
Biomedical Research Center, Osaka University, Medical School
-
TSUTSUI Tateki
Biomedical Research Center, Osaka University, Medical School
-
YOSHIDA Ryotaro
0saka Bioscience Institute
-
HERRMANN Steven
Genetics Institute
-
MU Jie
Department of Oncology, Biomedical Research Center
-
Hamaoka Toshiyuki
Department Of Oncology (c6) Osaka University Graduate School Of Medicine
-
Hamaoka Toshiyuki
Biomedical Research Center Faculty Of Medicine Osaka University
-
Hamaoka Toshiyuki
Biomedical Research Center Osaka University Medical School
-
Zou Jian-ping
Biomedical Research Center Osaka University Medical School
-
Fujiwara Hiromi
Biomedical Research Center Osaka University Medical School
-
Mu Jie
Department Of Oncology Biomedical Research Center
-
Fujiwara H
Department Of Oncology (c6) Graduate School Of Medicine Osaka University
-
Fujiwara Hiromi
Department Of Oncology Osaka University Graduate School Of Medicine
-
Takenaka Hiroshi
Biomedical Research Center Osaka University Medical School
-
Tsutsui Tateki
Department Of Oncology Biomedical Research Center
-
Hamaoka Toshiyuki
Department Of Oncology Osaka University Graduate School Of Medicine
-
Ogawa Makoto
Biomedical Research Center Osaka University Medical School
-
Yamamoto Norihiko
Biomedical Research Center Osaka University Medical School
-
Fujiwara H
Department Of Oncology Biomedical Research Center Osaka University Graduate School Of Medicine
-
Wijesuriya Rishani
Department Of Oncology Biomedical Research Center Osaka University Graduate School Of Medicine
関連論文
- SCID (Severe Combined Immunodeficiency) Mice as a New System to Investigate Metastasis of Human Tumors
- A mechanism underlying STAT4-mediated up-regulation of IFN-γ induction in TCR-triggered T cells
- Reversible CD8 expression induced by common cytokine receptor γchain-dependent cytokines in a cloned CD4^+ T_h1 cell line
- Requirement of IFN-γ in IL-12 production induced by collaboration between V_α14^+ NKT cells and antigen-presenting cells
- CD28 co-stimulatory signals induce IL-2 receptor expression on antigen-stimulated virgin T cells by an IL-2-independent mechanism
- B7.2-Ig fusion proteins enhance IL-4-dependent differentiation of tumor-sensitized CD8^+ cytotoxic T lymphocyte precursors
- The capacity of the natural ligands for CD28 to drive IL-4 expression in naive and antigen-primed CD4^+ and CD8^+ T cells
- Histiocytoid breast carcinoma : Solid variant of invasive lobular carcinoma with decreased expression of both E-cadherin and CD44 epithelial variant
- Differential IL-12 responsiveness of T cells but not of NK cells from tumor-bearing mice in IL-12-respomsive versus-unresponsive tumor models
- MOLECULAR MECHANISMS UNDERLYING T CELL MIGRATION RESPONSIBLE FOR IL-12-INDUCED TUMOR REGRESSION