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Department of Chemical Pharmacology, Faculty of Pharmaceutical Sciences, Toyama Medical and Pharmaceutical University | 論文
- Succinylcholine-Induced Acceleration and Suppression of Electrically Evoked Acetylcholine Release from Mouse Phrenic Nerve-Hemidiaphragm Muscle Preparation.
- Tetrodotoxin sensitivity of high K+-induced 86Rb efflux in the duodenal circular muscle of pigs.
- Alloxan-diabetic state-induced suppression of Ca2+-dependent slow action potentials in mouse diaphragm muscle.
- Identification and Characterization of a Novel Mutation M579Fs+75X in HERG
- Diabetes Mellitus-Induced Enhancement of Prostaglandin F2.ALPHA.-Responses Is Inhibited by Lipoxygenase- but Not Cyclooxygenase-Inhibitors in Mesenteric Veins and Arteries of Mouse and Rat.
- Inhibitory Effects of Magnoshinin and Magnosalin, Compounds from "Shin-i" (Flos Magnoliae), on the Competence and Progression Phases in Proliferation of Subcultured Rat Aortic Endothelial Cells.
- Dependence of Cholecystokinin-8-Stimulated Insulin Release on High Glucose Levels Is Evidenced by Pseudo-.ALPHA.-D-Glucose in Rat Pancreas and Islets.
- Heparin Inhibits the Progression Phase of Subcultured Endothelial Cell Proliferation in Rat Aorta.
- Different Modes of Potentiation by .BETA.-Eudesmol, a Main Compound from Atractylodes Lancea, Depending on Neuromuscular Blocking Actions of p-Phenylene-Polymethylene Bis-Ammonium Derivatives in Isolated Phrenic Nerve-Diaphragm Muscles of Normal and Allox
- Streptozocin-Diabetes Modifies Acetylcholine Release from Mouse Phrenic Nerve Terminal and Presynaptic Sensitivity to Succinylcholine.
- Increase in basal pulse rate and blood pressure by the diabetic state in KK-CAy mice, alloxan-mice and streptozotocin-mice.
- Modification of KCNH2-Encoded Cardiac Potassium Channels by KCNE1 Polymorphism:– Reply –