Kawabata Atsufumi | Division of Pharmacology & Pathophysiology, Kinki University School of Pharmacy, Japan

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著作論文

Physiology and Pathophysiology of Proteinase-Activated Receptors (PARs) : PAR-2 as a Potential Therapeutic Target
Modulation of Capsaicin-Evoked Visceral Pain and Referred Hyperalgesia by Protease-Activated Receptors 1 and 2
Proteinase-Activated Receptor-2-Triggered Prostaglandin E_2 Release, but Not Cyclooxygenase-2 Upregulation, Requires Activation of the Phosphatidylinositol 3-Kinase / Akt / Nuclear Factor-κB Pathway in Human Alveolar Epithelial Cells
Basic and Translational Research on Proteinase-Activated Receptors: Preface
Hydrogen Sulfide Causes Relaxation in Mouse Bronchial Smooth Muscle
Curcumin Inhibits the Proteinase-Activated Receptor-2–Triggered Prostaglandin E2 Production by Suppressing Cyclooxygenase-2 Upregulation and Akt-Dependent Activation of Nuclear Factor-κB in Human Lung Epithelial Cells
Physiology and Pathophysiology of Proteinase-Activated Receptors (PARs) : PARs in the Respiratory System : Cellular Signaling and Physiological/Pathological Roles
Curcumin Inhibits the Proteinase-Activated Receptor-2-Triggered Prostaglandin E_2 Production by Suppressing Cyclooxygenase-2 Upregulation and Akt-Dependent Activation of Nuclear Factor-κB in Human Lung Epithelial Cells
Colonic Hydrogen Sulfide-Induced Visceral Pain and Referred Hyperalgesia Involve Activation of Both Ca_v3.2 and TRPA1 Channels in Mice
Antihyperalgesic Effect of Buprenorphine Involves Nociceptin/Orphanin FQ Peptide–Receptor Activation in Rats With Spinal Nerve Injury–Induced Neuropathy

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