Takeda Toashihiro | Department Of Cardiovascular Medicine Osaka University Graduate School Of Medicine

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概要

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著作論文

FRS-071 Constitutive Autophagy Is Required to Maintain Cardiac Structure and Function in Aged Mouse Heart(FRS15,Novel Molecular Mechanisms of Heart Failure 1 (M),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japanese Circul
FRS-061 Cardiac-specific Disruption of Calpain4 Revealed Its Cardioprotective Role in Response to Pressure Overload(FRS13,Novel Molecular Mechanisms of Heart Failure 2 (M),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japan
PE-334 IKKβ/NF-κB Pathway Protects Hearts from Hemodynamic Stress Mediated through Regulating MnSOD Expression(Heart failure, basic(03)(M),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OE-255 Cross-talk between p38 Dependent Pathological Remodeling Cascade and Cardiac Adaptive Responses(Cardiac hypertrophy, basic/clinical(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
Presenilin 2 Regurates Excitation-contraction Coupling in Hearts(Excitation-contraction Coupling/Ion Channel 1 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OJ-525 Targeted Deletion of Apoptosis Signal-regulating Kinase 1 Attenuates Left Ventricular Remodeling(Molecular Biology, Myocardium 2 (M) : OJ64)(Oral Presentation (Japanese))
OJ-524 The Protective Role of STAT6 in Response to Pressure Overload in Vivo(Molecular Biology, Myocardium 2 (M) : OJ64)(Oral Presentation (Japanese))
OJ-023 Cardiac-specific Over-expression of Sarcolipin Inhibits the Sarcoplasmic Reticulum Ca^ ATPase (SERCA2a) and Reduces the Cardiac Function in Mice(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
OJ-020 Presenilin 2 Directly Interacts with Cardiac Ryanodine Receptor(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
PE-433 Reduction in Hemoglobin-Oxygen Affinity Results in the Improvement of Exercise Capacity in Mice with Chronic Heart Failure(Heart failure, basic-4, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-046 ASK1-p38 MAP Kinase Pathway Plays an Important Role in Physiological Hypertrophy Induced by Exercise Training(Cardiac hypertrophy, basic/clinical-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-066 Autophagy in Cardiomyocyte is Essential for Maintenance of Cardiac Function(Heart Failure, Cardiac Remodeling and Cardiomyopathy 1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-068 Cardiac-specific Activation of Apoptosis Signal-regulating Kinase 1 Leads to Cardiac Dysfunction through both Apoptotic and Non-apoptotic Cell Death(Cell death, apoptosis/necrosis (M) OE12,Oral Presentation (English),The 70th Anniversary Annual Sci
OE-067 Non-apoptotic Cell Death in Cardiac Injury : The Role of Cyclophilin D(Cell death, apoptosis/necrosis (M) OE12,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
FRS-015 Genetic Modulation of Ca^ Uptake Sarcoplasmic Reticulum Does Not Affect Maximal Ca^ Content and Ca^ Leakage in Mouse Myocardium(Cardiac Function "Ventricular Mechanics and Calcium Handling", The 71st Annual Scientific Meeting of the Ja
OJ-296 Overexpression of SERCA2a and Sarcolipin Does Not Affect Maximal Ca^ Content and Ca^ Leakage of Sarcoplasmic Reticulum in Mouse Myocardium(Cardiac function, basic/clinical-2 (M) OJ50,Oral Presentation (Japanese),The 70th Anniversary Annual
FRS-075 Chronic Inhibition of Apoptosis Signal-regulating Kinase 1 (ASK1) by Myocardial Gene Transfer Suppressed Progression of Heart Failure in Genetic Cardiomyopathy(Heart Failure Research (M) FRS15,Featured Research Session,The 70th Anniversary Annual
Involvement of Activation of ASK1 and NF-κB in the δ3 Isoform of Ca^/calmodulin-dependent Protein Kinase II Induced Cardiac Myocyte Hypertrophy(Cardiac Hypertrophy 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
p38α is Involved in the Mechanism of β-adrenergic Stress-induced Cardiomyocyte Cell Death and Cardiac Dysfunction(Molecular Biology, Myocardium 1 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OE-077 Downregulation of Ferritin Heavy Chain is an Important Trigger of Cardiac Cell Death and Progression of Heart Failure(Heart failure, basic(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)

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