Matsumura Yasushi | Department of Cardiology, Osaka University School of Medicine
スポンサーリンク
概要
関連著者
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Matsumura Yasushi
Department of Cardiology, Osaka University School of Medicine
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Matsumura Yasushi
Department Of Cardiology Osaka University School Of Medicine
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Matumura Yasushi
Ishinkai Yao General Hospital
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Matshumura Yasushi
Department Of Meddical Information Science Osaka University Graduate School Of Medicine
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Yamaguchi Osamu
Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine
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Takeda Toshihiro
Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine
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Nishida Kazuhiko
Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine
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Otsu Kinya
Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine
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Otsu Kinya
Department Of Cardiovascular Medicine
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Takeda Toshihiro
Department Of Cardiology Osaka University School Of Medicine
著作論文
- FRS-061 Cardiac-specific Disruption of Calpain4 Revealed Its Cardioprotective Role in Response to Pressure Overload(FRS13,Novel Molecular Mechanisms of Heart Failure 2 (M),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japan
- PE-334 IKKβ/NF-κB Pathway Protects Hearts from Hemodynamic Stress Mediated through Regulating MnSOD Expression(Heart failure, basic(03)(M),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-255 Cross-talk between p38 Dependent Pathological Remodeling Cascade and Cardiac Adaptive Responses(Cardiac hypertrophy, basic/clinical(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- Presenilin 2 Regurates Excitation-contraction Coupling in Hearts(Excitation-contraction Coupling/Ion Channel 1 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Cardiac-Specific Overexpression of a High Ca^ Affinity Mutant of Cardiac Ca^ ATPase Attenuates in Vivo Pressure Overload Cardiac Hypertrophy
- The Small G Protein Rac1 Induce Cardiac Myocyte Hypertrophy through the Activation of Apoptosis Signal-Regulating Kinase1 and Nuclear Factor-Kappa B
- Cardiac-Specific Disruption of Raf-1 Gene Induces Apoptosis and Cardiac Dysfunction
- The Antioxidant N-2-Mercaptopropionyl Glycine Attenuates Left Ventricular Hypertrophy in Murine Pressure-Overloaded Model
- Disruption of a single copy of the p38alpha mitogen-activated protein kinase gene leads to cardioprotection against ischemia-reperfision
- Amino acids ^KDDKPV^ in the cytosolic domain of SERCA2a are critical for functional association with PLN in vivo