Kusakari Yoichiro | Department of physiology (II), The Jikei University School of Medicine

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概要

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著作論文

Presenilin 2 Regurates Excitation-contraction Coupling in Hearts(Excitation-contraction Coupling/Ion Channel 1 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OJ-023 Cardiac-specific Over-expression of Sarcolipin Inhibits the Sarcoplasmic Reticulum Ca^ ATPase (SERCA2a) and Reduces the Cardiac Function in Mice(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
Beta-Adrenergic Stimulation Failed to Regulate Intracellular Ca^ Concentration and Isometric Tension in SERCA2a Overexpressing Heart
Cardiac-Specific Overexpression of a High Ca^ Affinity Mutant of Cardiac Ca^ ATPase Attenuates in Vivo Pressure Overload Cardiac Hypertrophy
FRS-015 Genetic Modulation of Ca^ Uptake Sarcoplasmic Reticulum Does Not Affect Maximal Ca^ Content and Ca^ Leakage in Mouse Myocardium(Cardiac Function "Ventricular Mechanics and Calcium Handling", The 71st Annual Scientific Meeting of the Ja
Intracellular Mechanism of the Negative Inotropic Effect Induced by α_1-Adrenoceptor Stimulation in Mouse Myocardium
Use of the Ca-Shortening Curve to Estimate the Myofilament Responsiveness to Ca^ in Tetanized Rat Ventricular Myocytes
OJ-296 Overexpression of SERCA2a and Sarcolipin Does Not Affect Maximal Ca^ Content and Ca^ Leakage of Sarcoplasmic Reticulum in Mouse Myocardium(Cardiac function, basic/clinical-2 (M) OJ50,Oral Presentation (Japanese),The 70th Anniversary Annual
A Directly Measurement of SR Ca^ Content in Mouse Ventricular Muscle : Not Vesicular SR(Cardiac Function, Basic/Clinical 1 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PJ-404 PKC signaling pathway is involved in negative inotropic effect and decrease in Ca^ sensitivity during α_1-adrenoceptor stimulation in mouse myocardium(Cardiac Function, Basic/Clinical 2 (M) : PJ68)(Poster Session (Japanese))
Characterization of Intracellular Ca^ Transient by the Hybrid Logistic Function in Aequorin-Injected Rabbit and Mouse Papillary Muscles
Superior Logistic Model for Decay of Ca2+ Transient and Isometric Relaxation Force Curve in Rabbit and Mouse Papillary Muscles
Effects of adrenergic stimulation on the Ca^ transients and tension in mouse left ventricular papillary muscles
The Mechanism of Increasing Ca^ Responsiveness by α_1-Adrenoceptor Stimulation in Rat Ventricular Myocytes
Use of Tetanus to Investigate Myofibrillar Responsiveness to Ca^ in Isolated Mouse Ventricular Myocytes

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