Imamura Hiroji | Department Of Thoracic And Cardiovascular Surgery Kansai Medical University Hirakata Hospital

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概要

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著作論文

Granulocyte-Colony Stimulating Factor Increases Donor Mesenchymal Stem Cells in Bone Marrow and Their Mobilization Into Peripheral Circulation but Does Not Repair Dystrophic Heart After Bone Marrow Transplantation
PJ-727 Inhibition of iNOS Uncoupling by Tetrahydrobiopterin Restores Tolerance to Ischemia/Reperfusion Injury in Streptozotocin-Induced Diabetic Rat Heart(Myocardial ischemia / reperfusion, basic / clinical(04)(IHD),Poster Session(Japanese),The 72nd Annua
OJ-182 The Mechanism of Oxidative Stress-induced Refractoriness to Ischemic Preconditioning in Aged Heart(Myocardial ischemia / reperfusion, basic / clinical(02)(IHD),Oral Presentation (Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulat
OE-308 Sarcolemmal Dystrophin is an End-Target of RISK Activation Mediated by Ischemic Postconditioning against Myocardial Infarction(Myocardial ischemia/reperfusion, basic/clinical(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting of
PE-564 Comparison of Safety of Mesenchymal Stem Cell Transplantation between Intracoronary Infusion and Direct Injection in Rats with Myocardial Infarction(Regeneration (angiogenesis/myocardial regeneration)-3, The 71st Annual Scientific Meeting of the Ja
PE-534 ATP-sensitive Redistribution of Utrophin : a Novel Mechanism of Cell Death during Metabolic Inhibition(Molecular biology, myocardium-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Role of Mechanical Stress in the Form of Cardiomyocyte Death During the Early Phase of Reperfusion
PJ-441 Angiotensinll Type-1 Receptor Blocker Preserves Ischemic Tolerance in Hypertensive Rat Heart by Converting Cardioprotective Effect from iNOS- to eNOS-dependent Mechanism(Acute myocardial infarction, basic-4 (IHD) 74,Poster Session (Japanese),The 70
PJ-106 Is Myocardial Hibernation a Self-defense Mechanism Against Contractile Force-induced Oncosis in Energy-depleted Myocyte?(Myocardial ischemia-reperfusion, basic/clinical-5 (IHD) PJ18,Poster Session (Japanese),The 70th Anniversary Annual Scientific M
PJ-105 Activation of Mitochondrial K_ Channels and Inhibition of Glycogen Synthase Kinase-3β Independently Contribute to Cardioprotection Mediated by Ischemic Preconditioning(Myocardial ischemia-reperfusion, basic/clinical-5 (IHD) PJ18,Poster Session
OJ-323 Regular Exercise Promotes Chronic Ischemic Tolerance via Activation of Cardiac Sympathetic Nerve, Oxidative Stress and Upregulation of iNOS(Acute myocardial infarction, basic-2 (IHD) OJ54,Oral Presentation (Japanese),The 70th Anniversary Annual Sci
OJ-246 Aged Heart is Tolerant to Ischemia/Reperfusion Injury by Exposure to Oxidative-Nitrosative Stress and Resistant to Ischemic Preconditioning(Myocardial ischemia-reperfusion, basic/clinical-2 (M) OJ41,Oral Presentation (Japanese),The 70th Anniversary
OJ-228 Intra-bone Marrow Transplantation of Bone Marrow Does Not Repair Cardiomyopathic Heart with Targeted Mutation of δ-sarcoglycan Gene(Cardiomyopathy, basic/clinical-2 (M) OJ38,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meetin
OE-028 Phosphorylation of Dystrophin is a Molecular Link between Mitochondrial Protection and Prevention of Oncosis Mediated by Ischemic Preconditioning(Myocardial ischemia-reperfusion, basic/clinical-1 (IHD) OE5,Oral Presentation (English),The 70th Anniv
Evaluation of chest computed tomography in patients after pneumonectomy to predict contralateral pneumothorax
Preoperative steroid therapy stabilizes postoperative respiratory conditions in myasthenia gravis
Early postoperative mobilization with walking at 4 hours after lobectomy in lung cancer patients
OJ-186 Role of Phosphatidylinositol 3-kinase-mTOR Cascade in Self-renewal and Myogenic Differentiation of Bone Marrow-derived Mesenchymal Stem Cells(Regeneration (angionenesis/myocardial regeneration)-3 (M) OJ31,Oral Presentation (Japanese),The 70th Anniv
FRS-057 IGF-1 Increases Engraftment of Transplanted Mesenchymal Stem Cells and Improves Cardiac Function without Differentiation in Rats with Myocardial Infarction(Regeneration Research in Cardiovascular Medicine (M) FRS12,Featured Research Session,The 70
Reintroduction of Contractile Force during Reperfusion Converts Myocyte Apoptosis to Necrosis (Acute Myocardial Infarction, Basic 5 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Dystrophin is an End-Target of Cardioprotection Mediated by Ischemic Preconditioning(Myocardial Ischemia-reperfusion, Basic 2 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Inhibition of p38 MAP kinase Ameliorates Heart Failure but Dissipates Ischemic Tolerance in Cardiomyopathic Heart(Acute Myocardial Infarction, Basic 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Regular Exercise Promotes Ischemic Tolerance by Upregulating iNOS(Acute Myocardial Infarction, Basic 1 (IHD)The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Bilaterally repeated spontaneous pneumothorax with ankylosing spondylitis

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