Moriguchi Akira | Cardiovascular Division Department Of Medicine 2 Kansai Medical University

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著作論文

DPJ-015 Urinary Liver-type Fatty-acid-binding Protein (L-FABP) could be an Early Predictive Marker for Impaired Renal Function after Coronary Angiography(DPJ03,Kidney/Renal Circulation/CKD (H),Digital Poster Session (Japanese),The 73rd Annual Scientific M
PJ-727 Inhibition of iNOS Uncoupling by Tetrahydrobiopterin Restores Tolerance to Ischemia/Reperfusion Injury in Streptozotocin-Induced Diabetic Rat Heart(Myocardial ischemia / reperfusion, basic / clinical(04)(IHD),Poster Session(Japanese),The 72nd Annua
OJ-182 The Mechanism of Oxidative Stress-induced Refractoriness to Ischemic Preconditioning in Aged Heart(Myocardial ischemia / reperfusion, basic / clinical(02)(IHD),Oral Presentation (Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulat
OE-308 Sarcolemmal Dystrophin is an End-Target of RISK Activation Mediated by Ischemic Postconditioning against Myocardial Infarction(Myocardial ischemia/reperfusion, basic/clinical(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting of
PE-564 Comparison of Safety of Mesenchymal Stem Cell Transplantation between Intracoronary Infusion and Direct Injection in Rats with Myocardial Infarction(Regeneration (angiogenesis/myocardial regeneration)-3, The 71st Annual Scientific Meeting of the Ja
PE-534 ATP-sensitive Redistribution of Utrophin : a Novel Mechanism of Cell Death during Metabolic Inhibition(Molecular biology, myocardium-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OJ-261 Carperitide can Prevent Acute Renal Failure from Contrast-media in Patients with Moderate Chronic Kidney Disease(OJ44,Kidney/Renal Circulation/CKD 2 (H),Oral Presentation (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Soc
PJ-146 Liver-type Fatty acid-binding protein is a promising predictive marker of contrast-induced nephropathy(Kidney/Renal circulation(05)(H),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
Inhibition of Contractile Activity During Postconditioning Enhances Cardioprotection by Restoring Sarcolemmal Dystrophin Through Phosphatidylinositol 3-Kinase
Signal Transduction Mechanism in Ischemic Preconditioning

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