Kyoi Shiori | The Second Department Of Internal Medicine Kansai Medical University

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著作論文

OJ-247 Antioxidant Paradox and Angiotensin II Type-1 Receptor Blockade for Heart Failure Treatment in Cardiomyopathy Hamster(Heart failure, basic-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Role of Mechanical Stress in the Form of Cardiomyocyte Death During the Early Phase of Reperfusion
PJ-156 Exercise-induced Activation of Cardiac Sympathetic Nerve Triggers Cardioprotection against Infarction via Redox-sensitive Activation of eNOS and Up-regulation of iNOS(Acute myocardial infarction, basic-2, The 71st Annual Scientific Meeting of the J
PJ-441 Angiotensinll Type-1 Receptor Blocker Preserves Ischemic Tolerance in Hypertensive Rat Heart by Converting Cardioprotective Effect from iNOS- to eNOS-dependent Mechanism(Acute myocardial infarction, basic-4 (IHD) 74,Poster Session (Japanese),The 70
PJ-106 Is Myocardial Hibernation a Self-defense Mechanism Against Contractile Force-induced Oncosis in Energy-depleted Myocyte?(Myocardial ischemia-reperfusion, basic/clinical-5 (IHD) PJ18,Poster Session (Japanese),The 70th Anniversary Annual Scientific M
PJ-105 Activation of Mitochondrial K_ Channels and Inhibition of Glycogen Synthase Kinase-3β Independently Contribute to Cardioprotection Mediated by Ischemic Preconditioning(Myocardial ischemia-reperfusion, basic/clinical-5 (IHD) PJ18,Poster Session
OJ-323 Regular Exercise Promotes Chronic Ischemic Tolerance via Activation of Cardiac Sympathetic Nerve, Oxidative Stress and Upregulation of iNOS(Acute myocardial infarction, basic-2 (IHD) OJ54,Oral Presentation (Japanese),The 70th Anniversary Annual Sci
OJ-246 Aged Heart is Tolerant to Ischemia/Reperfusion Injury by Exposure to Oxidative-Nitrosative Stress and Resistant to Ischemic Preconditioning(Myocardial ischemia-reperfusion, basic/clinical-2 (M) OJ41,Oral Presentation (Japanese),The 70th Anniversary
OE-028 Phosphorylation of Dystrophin is a Molecular Link between Mitochondrial Protection and Prevention of Oncosis Mediated by Ischemic Preconditioning(Myocardial ischemia-reperfusion, basic/clinical-1 (IHD) OE5,Oral Presentation (English),The 70th Anniv
Effect of Statin Therapy on In-stent Neointimal Proliferation after Coronary Stent Implantation ; 3D-Volumetric Intravascular Ultrasound Study (Restenosis after Angioplasty, Basic/Clinical 6 (IHD), The 69th Annual Scientific Meeting of the Japanese Circul
Relationship between Effectiveness of Statin Therapy on In-stent Restenosis and Arterial Remodeling in Patients with Acute Coronary Syndrome ; 3D-IVUS Study(Coronary Revascularization, PTCA/Stent/DCA/Rotablator/New Device 2 (IHD), The 69th Annual Scientif
Is It Appropriate to Implant Additional Drug Eluting Stent in the Moderate Lesion of the Vessel Subjected to Coronary Intervention?(Coronary Revascularization, PTCA/Stent/DCA/Rotablator/New Device 1 (IHD), The 69th Annual Scientific Meeting of the Japanes
Atorvastatin Prevents In-stent Restenosis after Coronary Stenting in Patients with Acute Coronary Syndrome(Coronary Revascularization, PTCA/Stent/DCA/Rotablator/New Device 2 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OJ-186 Role of Phosphatidylinositol 3-kinase-mTOR Cascade in Self-renewal and Myogenic Differentiation of Bone Marrow-derived Mesenchymal Stem Cells(Regeneration (angionenesis/myocardial regeneration)-3 (M) OJ31,Oral Presentation (Japanese),The 70th Anniv
FRS-057 IGF-1 Increases Engraftment of Transplanted Mesenchymal Stem Cells and Improves Cardiac Function without Differentiation in Rats with Myocardial Infarction(Regeneration Research in Cardiovascular Medicine (M) FRS12,Featured Research Session,The 70
Reintroduction of Contractile Force during Reperfusion Converts Myocyte Apoptosis to Necrosis (Acute Myocardial Infarction, Basic 5 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Dystrophin is an End-Target of Cardioprotection Mediated by Ischemic Preconditioning(Myocardial Ischemia-reperfusion, Basic 2 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Inhibition of p38 MAP kinase Ameliorates Heart Failure but Dissipates Ischemic Tolerance in Cardiomyopathic Heart(Acute Myocardial Infarction, Basic 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Regular Exercise Promotes Ischemic Tolerance by Upregulating iNOS(Acute Myocardial Infarction, Basic 1 (IHD)The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Inhibition of Contractile Activity During Postconditioning Enhances Cardioprotection by Restoring Sarcolemmal Dystrophin Through Phosphatidylinositol 3-Kinase

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