Miyauchi Hideyuki | Department of Cardiovascular Science and Medicine, Chiba University Graduate School of Medicine

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概要

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著作論文

1 p53-induced Inhibition of Angiogenesls Causes Cardiac Dysfunction during Pressure Overload(The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PE-538 Amlodipine Inhibits Vascular Cell Senescence and Protects against Atherogenesis through the Mechanism Independent of Calcium Channel Blockade(Molecular biology, vascular-3, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PE-412 Activation of SIRT1, a Longevity Gene, Prevents the Development of Diabetic Vasculopathy by Inhibiting Endothelial Senescence(Diabetes/Obesity/Metabolic syndrome-05, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-204 A Protective Role of Brain-Derived Neurotrophic Factor in Cardiac Remodeling after Myocardial Infarction(Cardiac function, basic/clinical-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-007 Hyperinsulinemia Induces Endothelial Senescence and Promotes the Development of Diabetic Vasculopathy via a p21-dependent Pathway(Molecular biology, vascular-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PJ-149 Hyperglycemia-induced Senescence of Endothelial Cells through the p53 Deacetylase SIRT1(Diabetes/Obesity/Metabolic syndrome-10 (H) PJ25,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
OE-105 Granulocyte-colony stimulating factor prevents transition from left ventricular hypertrophy to diastolic heart failure in hypertensive rats(Heart Failure, Basic 1 (M) : OE13)(Oral Presentation (English))
PE-398 Molecular Mechanism of Non-dipper Hypertension Associated with Aging(Hypertension, basic-2 (H) PE67,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Mechanisms of Impaired Circadian Rhythm Associated with Cellular Senescence (Molecular Biology, Vascular 2 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Role of Angiotensin II-induced Vascular Cell Senescence in the Progression of Atherosclerosis(Atherosclerosis, Basic 8 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Insulin-induced Endothelial Cell Senescence Causes Diabetic Vasculopathy(Diabetes/Obesity 5 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Akt negatively regulates the in vitro lifespan of human endothelial Cells via a p53/p21-dependent pathway
OJ-519 Telomerase restores impaired circadian rhythm associated with cellular senescence(Vascular Smooth Muscle 2 (H) : OJ63)(Oral Presentation (Japanese))
Akt Induces Endothelial Cell Senescence via a p53/p21-Dependent Pathway
Peripheral Mononuclear Cell Implantation Is Effective to Patients with Peripheral Artery Disease
Introduction of telomerase restores endothelial dysfunction associated with aging
Pathophysiological role of telomerase activation in vascular remodeling
Vascular smooth muscle cell senescence promotes vascular inflammation in human atherosclerosis
OE-207 Granulocyte-Colony Stimulating Factor Stimulates Preexisting Endothelial Cells and Promotes Blood Vessel Formation in vitro(Angiogenesis 1 (M) : OE26)(Oral Presentation (English))

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