Akazawa Hiroshi | Department Of Cardiovascular Science And Medicine Chiba University

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著作論文

DPE-008 Bone Marrow Derived Mononuclear Cells Improve the Cardiac Function of Dilated Cardiomyopathy Models via Gr-1 Positive Cells-secreted Factors(DPE02,Heart Failure, Basic (M),Digital Poster Session (English),The 73rd Annual Scientific Meeting of The
PE-424 Bone Marrow Derived Monomuclear Cells Transiently Improve the Cardiac Function of Dilated Cardiomyophthy Models via Gr-1 Positive cells-secreted Factors(Heart failure, basic(04)(M),Poster Session(English),The 72nd Annual Scientific Meeting of the J
Multivalent ligand-receptor interactions elicit inverse agonist activity of AT_1 receptor blockers against stretch-induced AT_1 receptor activation
OE-164 Atg7 is Essential for Starvation-induced And Constitutive Autophagy in the Hearts(Molecular biology, myocardium (M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OE-020 Molecular and Structural Basis for Mechanical Stress-induced Activation of AT Receptor(Cardiovascular pharmacology, basic/clinical(01)(H),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
FRS-009 Crucial roles of PDK1 in the regulation of β-adrenergic response and cell survival in the hearts(Novel Pathophysiological Mechanisms of Heart Failure(M),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Socie
Angiotensin II Type 1a Receptor Signals are Involved in the Progression of Heart Failure in MLP-Deficient Mice
PE-178 Different Aspects of AT1 Receptor Blockers in Showing Inverse Agonist Activity(Cardiac hypertrophy, basic/clinical-3 (M) PE30,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
1 Actions on Angiotensin II Receptor by Angiotensin II, Mechanical Stress, And Inverse Agonists(Molecular Mechanism of Hypertensive Heart Failure,Symposium 5 (SY5) (H),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
FRS-006 Targeting p53 for therapy against myocardial infarction(Novel Pathophysiological Mechanisms of Heart Failure(M),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
Cardiomyocytes Fuse with Surrounding Non-Cardiomyocytes and Re-enter the Cell Cycle(The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PE-359 Cytokine Therapy Prevents Left Ventricular Remodeling And Dysfunction after Myocardial Infarction Through Neovascularization.(Cardiovascular Pharmacology, Basic/Clinical 4 (H) : PE61)(Poster Session (English))
PE-197 Adult Cardiac Sca-1 Positive Cells Differentiate into Beating Cardiomyocytes(Development and Differentiation 1 (M) : PE34)(Poster Session (English))
OE-368 Ligand-independent activation of angiotensin II type 1 receptor by mechanical stretch(Cardiac Hypertrophy, Basic and Clinical 1 (M) : OE46)(Oral Presentation (English))
OE-364 Exercise-induced upregulation of _1-adrenergic receptor is involved in the upregulation of Hsp70 through enhanced activity of ERKs(Cardiac Hypertrophy, Basic and Clinical 1 (M) : OE46)(Oral Presentation (English))
OE-105 Granulocyte-colony stimulating factor prevents transition from left ventricular hypertrophy to diastolic heart failure in hypertensive rats(Heart Failure, Basic 1 (M) : OE13)(Oral Presentation (English))
OE-004 Leukemia inhibitory factor enhances survival of cardiomyocytes and induces regeneration of myocardium after myocardial infarction(Acute Myocardial Infarction, Basic 1 (IHD) : OE1)(Oral Presentation (English))
2 Diphtheria toxin-induced autophagic death of cardiomyocytes plays a pathogenic role in mouse model of heart failure(Young Investigator's Award Finalists Lectures (YIA))(Special Program)
Effects of Torasemide on Heart Failure
Rat Neonatal Cardiomyocytes Fuse with Mammalian Somatic Cellsand Reenter Cell Cycle
Angiotensin II Type 1 Receptor May Act as a Mechanoceptor in Stretch-Induced Cardiomyocyte Hypertrophy
Heat Shock Proteins Are Upregulated in Exercised-lnduced Cardiac Hypertrophy But Not in Pressure Overload-Induced Cardiac Hypertrophy
FRS-069 PDK-1 Plays Essential Roles in Cell Size Regulation, Beta-adrenergic Response and Cell Survival in the Heart(Heart Failure, Cardiac Remodeling and Cardiomyopathy 1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-072 PDK-1 is Required for Regulation of Cell Size and Beta-adrenergic Response in the Hearts(Heart Failure Research (M) FRS15,Featured Research Session,The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
OJ-526 Targeted Expression of a Dominant-negative EGFR in the Heart Elicits an Essential Role of ErbB Signaling in Normal Cardiac Function(Molecular Biology, Myocardium 2 (M) : OJ64)(Oral Presentation (Japanese))
FRS-062 Calpastatin Deficiency Reveals Calpain-dependent Progression of Left Ventricular Remodeling after Myocardial Infarction(FRS13,Novel Molecular Mechanisms of Heart Failure 2 (M),Featured Research Session (English),The 73rd Annual Scientific Meeting
Targeted Expression of a Dominant-negative EGFR in the Heart Causes Progressive Heart Failure by Disrupting Mitochondrial Integrity(Clinical and Basic Research in Heart Failure (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OJ-068 Protective Roles of Heat Shock Transcription Factor-1 in Physiological and Pathological Cardiac Hypertrophy(Cardiac hypertrophy, basic/clinical-2 (M) OJ12,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese C
FRS-171 Role of Na+-Ca2+ Exchanger in Myocardial Ischemia/Reperfusion Injury : Evaluation Using a Heterozygous Na+-Ca2+ Exchanger Knockout Mouse Model(Coronary Heart Disease : Basic Science (IHD) : FRS20)(Featured Research Session (English))
OE-257 Mononuclear Cell Implantation Induces Neovascularization through Muscle Regeneration(Regeneration (angionenesis/myocardial regeneration)-1 (M) OE43,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulati
IS003 Transcriptional Regulation of Human Cardiac Homeobox Gene CSX1-Role of Bone Morphogenetic Proteins and Downstream Signaling Molecules
Cardiomyocyte Apoptosis Is a Cause of Heart Failure : A New Mouse Model of Heart Failure
Functional analysis of CAL, a Cofactor for the Homeobox Transcription Factor CSX / NKX-2.5
Diverse effects of homeobox transcription factor Csx/Nkx2-5 on cardiac myocytes
OJ-509 Gender Difference in Effect of AT1 Blockade on Murine DCM model(Cardiomyopathy, Basic (M) : OJ62)(Oral Presentation (Japanese))
Different gene expressions in volume overload and pressure overload hypertrophy
Cardioprotective genes are elevated in physiological hypertrophy
Targeted lacking of ryanodine receptor type 2 in the heart suppresses pressure overload-induced hypertrophic responses but impairs cardiac functions
Phosphatidylinositol 3-kinase Plays Critical Roles in Early Cardiomyogenesis by Regulating Wnt Signaling(Development and Differentiation 3 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PE-256 Olmesartan Ameliorates Experimental Heart Failure Induced by Diphtheria Toxin-mediated Cardiomyocyte Ablation(Neurohumoral Factors 2 (H) : PE44)(Poster Session (English))
Overexpression of Heat Shock Factor 1 in the heart protects cardiomyocytes from injury after ischemia/reperfusion
Molecular and Cellular Mechanisms of Mechanical Stress-Induced Cardiac Hypertrophy
Continuous Blockade of L-Type Ca^ Channels Suppresses Activation of Calcineurin nad Development of Cardiac Hypertrophy in Spontaneously Hypertensive Rats
Gene expression profile revealed different effects of ARB and ACE-I on heart failure
FRS-009 Close relationship between Wnt and PI3-kinase pathway during earliest cardiomyocyte differentiation(Angiogenesis and Regeneration Medicine in Cardiovascular Medicine (M) : FRS2)(Featured Research Session (English))

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