Moriya Junji | Department Of Cardiovascular Science And Medicine Chiba University Graduate School Of Medicine

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著作論文

1 p53-induced Inhibition of Angiogenesls Causes Cardiac Dysfunction during Pressure Overload(The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OJ-248 A Pathological Role of Semaphorin3E/PlexinD1 in Impaired Angiogenesis of Diabetes(OJ42,Peripheral Circulation/Vascular Disease (Pathophysiology, Basic) (H),Oral Presentation (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation
PJ-174 Semaphorin 3E/Plexin D1 as a Novel Target for Therapeutic Angiogenesis(Regeneration(angiogenesis/myocardial regeneration)(04)(M),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
4 In-vivo Monitoring of Manganese-labeled White Blood Cells for Regenerative Therapy in the Ischemic Rat(Role of Molecular Imaging for Cardiovascular Regeneration Treatments,Topic 2 (TP-2) (I),Special Program,The 72nd Annual Scientific Meeting of the Japa
PE-545 Atologous Transplantation of Peripheral Blood Mononuclear Cells for Patients with Critical Limb Ischemia : Long Term Outcomes(Peripheral circulation/Vascular disease-4, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PE-538 Amlodipine Inhibits Vascular Cell Senescence and Protects against Atherogenesis through the Mechanism Independent of Calcium Channel Blockade(Molecular biology, vascular-3, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PE-412 Activation of SIRT1, a Longevity Gene, Prevents the Development of Diabetic Vasculopathy by Inhibiting Endothelial Senescence(Diabetes/Obesity/Metabolic syndrome-05, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-204 A Protective Role of Brain-Derived Neurotrophic Factor in Cardiac Remodeling after Myocardial Infarction(Cardiac function, basic/clinical-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-007 Hyperinsulinemia Induces Endothelial Senescence and Promotes the Development of Diabetic Vasculopathy via a p21-dependent Pathway(Molecular biology, vascular-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)

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