OE-298 Eicosapentaenoic Acid, a Major Component of Fish Oil, Prevents the Progression of Endothelin-1-Induced Cardiomyocyte Hypertrophy through Suppressing Phosphorylated JNK(Cardiovascular pharmacology, basic/clinical-1 (H) OE50,Oral Presentation (Englis
スポンサーリンク
概要
- 論文の詳細を見る
- 社団法人日本循環器学会の論文
- 2006-03-01
著者
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Goto Katsutoshi
Departments of Internal Medicine, Surgery, Pharmacology, University of Tsukuba
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Masuoka T
Cardiology Department Of Internal Medicine University Of Tsukuba Graduate School Of Comprehensive Hu
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Shimojyoh Nobutake
Division of Cardiovascular Medicine, University of Tsukuba
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Miyauchi Takashi
Division of Cardiology, Department of Internal Medicine, University of Tsukuba
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Machino Takeshi
Cardiology Department Of Internal Medicine University Of Tsukuba Graduate School Of Comprehensive Hu
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Yamaguchi Iwao
Division of Cardiology, Graduate School of Comprehensive Human Sciences, University of Tsukuba
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Shimojyoh Nobutake
Cardiovascular Division Department Of Internal Medicine Institute Of Clinical Medicine University Of
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Zaedi Sohel
Division Of Gene Therapeutics Research Institute International Medical Center Of Japan
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Maeda Seiji
Institute Of Health And Sport Sciences Graduate School Of Comprehensive Human Science University Of
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Subrina Jesmin
Division Of Gene Therapeutics Research Institute International Medical Center Of Japan
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Subrina Jesmin
Center For Tsukuba Advanced Research Alliance (tara) University Of Tsukuba
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MAEDA Seiji
Division of Sports Medicine, Graduate School of Comprehensive Human Sciences, University of Tsukuba
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Jesmin Subrina
Division of Gene Therapeutics, Research Institute, International Medical Center of Japan
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Shimojo Nobutake
Division of Cardiovascular Medicine, University of Tsukuba
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Soma Masaaki
Division of Cardiovascular Medicine, University of Tsukuba
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Soma Masaaki
Division Of Cardiovascular Medicine University Of Tsukuba
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Goto Katsutoshi
Department Of Pharmacology University Of Tsukuba
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Goto Katsutoshi
Department Of Pharmacology Institute Of Basic Medical Scienses University Of Tsukuba
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Maeda Seiji
Division Of Sports Medicine Graduate School Of Comprehensive Human Sciences University Of Tsukuba
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Yamaguchi Iwao
Division Of Cardiology Institute Of Clinical Medicine Graduate School Of Comprehensive Human Science
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Goto Katsutoshi
Department Of Pharmacology Institute Of Basic Medical Sciences The University Of Tsukuba
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Yamaguchi Iwao
Department Of Cardiovascular Medicine Institute Of Clinical Medicine Universily Of Tsukuba
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Jesmin Subrina
Division Of Gene Therapeutics Research Institute International Medical Center Of Japan
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Shimojo Nobutake
Cardiovascular Division Institute Of Clinical Medicine Graduate School Of Comprehensive Human Scienc
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Jesmin Subrina
Center For Tsukuba Advanced Research Alliance (tara) University Of Tsukuba:institute Of Clinical Med
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Machino Takeshi
Cardiovascular Division Institute Of Clinical Medicine University Of Tsukuba
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Shimojo Nobutake
Cardiovascular Division Institute Of Clinical Medicine Graduate School Of Comprehensive Human Scienc
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Goto Katsutoshi
Department Of Ophthalmology Kawasaki Medical School
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- PJ-237 Utility of Septal Flash Motion as a Predictor of Responders to Cardiac Resynchronization Therapy(PJ040,CRT/ICD 2 (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
- PJ-449 Detection of 'Injury Current' of Ischemic Myocardium by Magnetocardiography : A Treadmill-Stress Magneteocardiographic Study(PJ076,Exercise Test/Cardiac Rehabilitation 1 (H),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japane
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- DPJ-021 Commercially Available Serum N-terminal proBNP Level Accurately Differentiates Presence of Cardiac Morphological and Hemodynamic Abnormalities Compared with Brain Natriuretic Peptide(DPJ04,Heart Failure (Diagnosis) (M),Digital Poster Session (Japa
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- PE-118 Preliminary Feasibility and Early Clinical Outcome of the Ablation of Culprit Veins That Drives Paroxysmal Atrial Fibrillation(Arrhythmia, therapy-09, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
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- PE-288 Elimination of Delayed Potential by Catheter Ablation Based on High Resolution Substrate Mapping of Reentrant Ventricular Tachyarrhythmia(Arrhythmia, therapy-13, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
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- PJ-777 Role of Endothelin in the Impairment Cardiac Function, Cardiac VEGF Signaling and Coronary Collateral Development in Early Diabetic Rats(Diabetes/Obesity/Metabolic syndrome-12, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-776 Normalization of Cardiac Upregulated Endothelin-1 System in Type 2 Diabetic Rat by A Long-Acting Calcium Channel Blocker at Sub-Depressor Dose(Diabetes/Obesity/Metabolic syndrome-12, The 71st Annual Scientific Meeting of the Japanese Circulation So
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- PJ-573 Chronic Treatment with Endothelin Receptor Antagonist Normalizes the Altered Cerebral Capillary Density in the Frontal Cortex of SHR-SP Rats(Cerebrovascular circulation/Stroke-3, The 71st Annual Scientific Meeting of the Japanese Circulation Societ
- PJ-556 Estrogen Pre-treatment Prevents the ET-1-induced Cardiomyocyte Hypertrophy in vitro : Potential Linkage with VEGF(Cardiovascular pharmacology, basic/clinical-5, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-538 VEGF Signaling is Disrupted in the Heart of Mice Lacking Estrogen Receptor(Neurohumoral factors-3, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-320 Endothelin Antagonism Greatly Reverses Upregulated Renin-Angiotensin System in the Hypertrophied Heart of SHR-SP(Cardiac hypertrophy, basic/clinical-4, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-254 'Injury Current' in Patients with coronary Stenosis Can be Visualized by Magnetocardiography, and Continues to Exist Early after Coronary Revascularization(Myocardial ischemia/reperfusion, basic/clinical-4, The 71st Annual Scientific Meeting of the
- PE-151 Detection of the 'Injury Current' of Ischemic Myocardium by High-Tc-SQUID Magnetocardiography(ECG/Body surface potential mapping/Holter-4, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
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- -0705- Endothelin-1 and cardiac hypertrophy due to pressure or volume overload in rats(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
- -0657- Effects of an endothelin-A receptor antagonist on the pulmonary hypertension caused by congestive heart failure in Rats(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
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- The Production of Endothelin-1 in the Hypertrophied Rat Heart due to Pressure or Volume Overload
- Orexin Receptor Type-1 Couples Exclusively to Pertussis Toxin-Insensitive G-Proteins, while Orexin Receptor Type-2 Couples to Both Pertussis Toxin-Sensitive and *Insensitive G-Proteins
- Bosentan, a novel synthetic mixed-type endothelin receptor antagonist, attenuates acute gastric mucosal lesions induced by indomethacin and HCl in the rat: Role of endogenous endothelin-1
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- Combination of endothelin-A (ET_A) receptor inhibitor and prostacyclin (PGI_2) analogue is preferable to each drug alone for pulmonary hypertension (PH)
- Corresponding distributions of increased endothelin-B receptor expression and increased endothelin-1 expression in the aorta of apolipoprotein E-deficient mice with advanced atherosclerosis