-P615- ANATOMICAL AND BIOCHEMICAL REGRESSION OF CARDIACHYPERTROPHY BY A LONG-ACTING CALCIUM ANTAGONIST AMLODIPINE(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)

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概要

• 論文の詳細を見る
• 社団法人日本循環器学会の論文
• 1995-06-20

著者

• Zou Y

  Department Of Cardiovascular Science And Medicine Chiba University Graduate School Of Medicine

• Kudoh Sumiyo

  Department Of Internal Medicine Iii Osaka Medical College

• Sohmiya Koichi

  Department Of Internal Medicine Iii Osaka Medical College

• Yamazaki Tsutoinu

  Department Of Clinical Epidemiology And Systems University Of Tokyo

• Shiojima Ichiro

  Department of Cardiovascular Science and Medicine, Chiba University Graduate School of Medicine

• Yunzeng Zou

  東京大学 第3内科

• Nagai Ryozo

  Third Department of Internal Medicine, Faculty of Medicine, University of Tokyo

• Yazaki Yoshio

  Third Department Of Internal Medicine Faculty Of Medicine University Of Tokyo

• YAMAZAKI TSUTOMU

  Third Department of Internal Medicine, Faculty of Medicine, University of Tokyo

• Yunzeng Zou

  Third Department of Internal Medicine, Faculty of Medicine, University of Tokyor

• Kudoh Sumiyo

  Third Department of Internal Medicine, Faculty of Medicine, University of Tokyor

• Shiojima Ichiro

  Third Department of Internal Medicine, Faculty of Medicine, University of Tokyor

• Hiroi Yukio

  Third Department of Internal Medicine, Faculty of Medicine, University of Tokyor

• Mizuno Takehiko

  Third Department of Internal Medicine, Faculty of Medicine, University of Tokyor

• Aikawa Ryuichi

  Third Department of Internal Medicine, Faculty of Medicine, University of Tokyor

• Takano Hiroyuki

  Third Department of Internal Medicine, Faculty of Medicine, University of Tokyor

• SHIOJIMA Ichiro

  3rd Dept. of Internal Medicine

• Aikawa Ryuichi

  Department Of Cardiovascular Medicine University Of Tokyo Graduate School Of Medicine

• Komuro Issei

  Third Department Of Internal Medicine Faculty Of Medicine University Of Tokyo

• Yazaki Yoshio

  The Third Dept. Of Medicine University Of Tokyo

• Shiojima Ichirou

  The Third Department Of Internal Medicine University Of Tokyo

• Yui Yoshiki

  京都大学 医研究 循病態

• Yamazaki Tsutomu

  Department Of Clinical Epidemiology And Systems University Of Tokyo

• Mizuno Takehiko

  Department Of Cardiocvascular Medicine University Of Tokyo Graduate School Of Medicine

• Mizuno Takehiko

  The Third Department Of Internal Medicine University Of Tokyo

• Nagai Ryozo

  Third Department Of Internal Medicine Faculty Of Medicine University Of Tokyo

• Yazaki Yoshio

  Third Department Of Internal Medicine Faculty Of Medicine The University Of Tokyo.

• Yamazaki Tsutomu

  Department Of Clinical Epidemiology & Systems The University Of Tokyo

関連論文

• Effects of Medication on Cardiovascular Events in the Japanese Coronary Artery Disease (JCAD) Study
• Left Atrial Dyssynchrony in Patients With Nonobstructive Hypertrophic Cardiomyopathy Evaluated by Myocardial Strain Imaging
• PE-330 Temporal Relationship of Myocardial Wall Strain Can Determine the State of Heart Failure in Patients Undergoing Right Ventricular Apical Pacing(Echo/Doppler-11, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• PE-328 Intraventricular Mechanical Dyssynchrony in Patients Undergoing Long-term Right Ventricular Apical Pacing : Relation to B-type Natriuretica Protein Levels(Echo/Doppler-11, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• Gitelman's Syndrome With Exercise-Induced Ventricular Tachycardia
• 1 p53-induced Inhibition of Angiogenesls Causes Cardiac Dysfunction during Pressure Overload(The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• PE-178 Different Aspects of AT1 Receptor Blockers in Showing Inverse Agonist Activity(Cardiac hypertrophy, basic/clinical-3 (M) PE30,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
• 1 Actions on Angiotensin II Receptor by Angiotensin II, Mechanical Stress, And Inverse Agonists(Molecular Mechanism of Hypertensive Heart Failure,Symposium 5 (SY5) (H),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
• Cytokine Therapy has Beneficial Effects on Cardiac Remodeling and Vascular Density after Acute Myocardial Infarction in Swine (Vasoactive Compound/Growth Factor/Cytokine 2 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Cardioprotective Effects of Granulocyte Colony-stimulating Factor in Chronic Hibernating Swine Myocardium(Old Myocardial Infarction/Remodeling 3 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Beneficial Effects of G-CSF on Coronary Atherosclerotic Lesion in Watanabe Heritable Hyperlipidemic Rabbits(Cardiovascular Pharmacology, Basic/Clinical 1 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• The Direct Effects of G-CSF on Cardioprotection against Ischemia/Reperfusion Injury(Myocardial Ischemia-reperfusion, Basic 3 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• PPARgamma Ligand Inhibits the Progression of Inflammation in Myosin-induced Autoimmune Myocarditis in Rats (Myocarditis, Basic/Clinical 1 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• OJ-482 G-CSF Prevents Cardiac Remodeling after Acute Myocardial Infarction in Swine(Old Myocardial Infarction/Remodeling 1 (IHD) : OJ59)(Oral Presentation (Japanese))
• Effects of Nicorandil on Cardiovascular Events in Patients With Coronary Artery Disease in The Japanese Coronary Artery Disease (JCAD) Study
• Gender Differences in Patients With Coronary Artery Disease in Japan : The Japanese Coronary Artery Disease Study (The JCAD Study)
• Elevated Serum Uric Acid is an Independent Predictor for Cardiovascular Events in Patients With Severe Coronary Artery Stenosis : Subanalysis of the Japanese Coronary Artery Disease (JCAD) Study
• OJ-160 Comparison of Risk Factors between Angiographically Diffuse versus Focal Stenosis in the Patients Enrolled in Japanese Coronary Artery Disease (JCAD) Study(OJ27,Coronary Circulation/Chronic Coronary Disease (Basic/Clinical) (IHD),Oral Presentation
• OE-390 Association between Angiographical Assessment of Luminal Narrowing and Recurrence of Cardiovascular Events : Substudy of Japanese Coronary Artery Disease (JCAD) Study(OE66,Atherosclerosis (Clinical/Pathophysiology) (IHD),Oral Presentation (English)
• FRS-087 The Impact of Diabetes on the Prognosis of Japanese Patients with Coronary Artery Disease(FRS18,New Insights into Metabolic Syndrome (H),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
• Elevated Serum C-Reactive Protein Levels Predict Cardiovascular Events in the Japanese Coronary Artery Disease (JCAD) Study(Epidemiology)
• Demographics and Changes in Medical/Interventional Treatment of Coronary Artery Disease Patients Over a 3.5-Year Period in Japan : The Japanese Coronary Artery Disease Study: Trend Examination
• Rationale and Design of a Study to Examine Lower Targets for Low-Density Lipoprotein-Cholesterol and Blood Pressure in Coronary Artery Disease Patients : The Japanese Coronary Artery Disease Study II (JCADII)
• OJ-217 Genetic Variations of Mrf-2/Arid5B Confers Risk of Coronary Atherosclerosis in the Japanese Population(Genetics/Genetically engineered models/Gene therapy-2(H/M), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• PJ-482 The Polymorphism in the Adiponectin Gene is Associated with Susceptibility to Coronary Artery Disease(Atherosclerosis, clinical-11 (H) PJ81,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society
• ATP binding Cassette Transporter (ABCA1) variant is one of the determinantsof plasma high density lipoprotein level in Japanese population
• PE-359 Cytokine Therapy Prevents Left Ventricular Remodeling And Dysfunction after Myocardial Infarction Through Neovascularization.(Cardiovascular Pharmacology, Basic/Clinical 4 (H) : PE61)(Poster Session (English))
• OE-368 Ligand-independent activation of angiotensin II type 1 receptor by mechanical stretch(Cardiac Hypertrophy, Basic and Clinical 1 (M) : OE46)(Oral Presentation (English))
• OE-364 Exercise-induced upregulation of _1-adrenergic receptor is involved in the upregulation of Hsp70 through enhanced activity of ERKs(Cardiac Hypertrophy, Basic and Clinical 1 (M) : OE46)(Oral Presentation (English))
• OE-105 Granulocyte-colony stimulating factor prevents transition from left ventricular hypertrophy to diastolic heart failure in hypertensive rats(Heart Failure, Basic 1 (M) : OE13)(Oral Presentation (English))
• OE-004 Leukemia inhibitory factor enhances survival of cardiomyocytes and induces regeneration of myocardium after myocardial infarction(Acute Myocardial Infarction, Basic 1 (IHD) : OE1)(Oral Presentation (English))
• 2 Diphtheria toxin-induced autophagic death of cardiomyocytes plays a pathogenic role in mouse model of heart failure(Young Investigator's Award Finalists Lectures (YIA))(Special Program)
• Effects of Torasemide on Heart Failure
• Rat Neonatal Cardiomyocytes Fuse with Mammalian Somatic Cellsand Reenter Cell Cycle
• Angiotensin II Type 1 Receptor May Act as a Mechanoceptor in Stretch-Induced Cardiomyocyte Hypertrophy
• Heat Shock Proteins Are Upregulated in Exercised-lnduced Cardiac Hypertrophy But Not in Pressure Overload-Induced Cardiac Hypertrophy
• PE-066 Tyrosine Kinase-Dependent Mechanism Occurs Downstream of Protein Kinase C Activation in the Pharmacological Preconditioning by Alpha-1 Adrenoceptor Stimulation(Acute Myocardial Infarction, Basic 3 (IHD) : PE12)(Poster Session (English))
• The Effect of Edaravone on Apoptotic Cell Formation during Ischemia-Reperfusion in Rabbit Heart
• Anti-Ischemic Effect of Alpha-Adrenergic Stimulation Is Mediated by Tyrosine Kinase-Dependent Mechanism
• Hypoxic Preconditioning Induces the Expression of Heme Oxygenase-1 via P38 Mitogen-Activated Protein Kinase (p38MAPK) Pathway in Cardiac Fibroblasts
• OJ-062 Effect of Chymase Inhibition by NK-3201 on Hypoxia-Induced Left Ventricular Remodeling in Mice(Cardiovascular pharmacology, basic/clinical(02)(H),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
• OE-245 Critical Role of Cardiac Angiogenesis in Regulating Cardiac Hypertrophy and Function(Cardiac hypertrophy, basic/clinical-1 (M) OE41,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
• Critical Role of Cardiac Angiogenesis in Regulating Cardiac Hypertrophy and Function(Cardiac Hypertrophy 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Targeted Expression of a Dominant-negative EGFR in the Heart Causes Progressive Heart Failure by Disrupting Mitochondrial Integrity(Clinical and Basic Research in Heart Failure (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Angiotensin-II Receptor Blocker Exerts Cardioprotection in Diabetic Rats Exposed to Hypoxia(Experimental Investigation)
• OE-012 Angiotensin II Receptor Blocker Inhibits NF-κ B Activity and Prevents Hypoxia-Induced Fibrosis of Left Ventricular Myocardium in Apolipoprotein E-Knockout Mice(Chronic coronary heart disease/Remodeling-1 (IHD) OE2,Oral Presentation (English),The 70
• Chronic Hypoxia Accelerates the Progression of Atherosclerosis in Apolipoprotein E-Knockout Mice
• Mechanisms of Combined Treatment With Celiprolol and Candesartan for Ventricular Remodeling in Experimental Heart Failure
• Nifedipine Decreases Oxidative Stress and Exhibits Cardiovascular Protection in Diabetic Rats Exposed to Hypoxia(Diabetes/Obesity 3 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• PE-322 Hypoxia Induces Atherogenesis of Apolipoprotein E-knockout Mice through Superoxide Production(Atherosclerosis, Basic 5 (IHD) : PE55)(Poster Session (English))
• OE-320 Angiotensin-II Receptor Blocker Exhibits Cardioprotection through Modulation of Cytokine Expression in the Metabolic Syndrome Accompanied with Hypoxia(Diabetes/Obesity 2 (H) : OE40)(Oral Presentation (English))
• Nifedipine Enhances Cardioprotective Effect of Angiotensin-II Receptor Blocker in Progressive Heart Failure
• Endothelin ETA Receptor Antagonist Suppressed ET-1 Content and INOS but Could Not Improve the Cardiac Dysfunction in Dilated Cardiomyopathy
• New Concept of Cardioprotection in Diabetes Mellitus from a Viewpoint of Intracellular Free-Thiol Stabilization by Ebselen
• Altered pattern of gene expression in heart failure
• PE-425 Left Ventricular Remodeling Induced by Intermittent Hypoxia : Role of gp91phox Subunit of NADPH Oxidase(Heart failure, basic(04)(M),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
• PE-398 Nuclear Factor-κ B Activation Accelerates Cardiac Hypertrophy, Interstitial Fibrosis, and Oxidative Stress in Diabetic Rats Exposed to Intermittent Hypoxia(Chronic coronary heart disease/Remodeling-2, The 71st Annual Scientific Meeting of the Japan
• Two Distinct Mechanisms of Angiotensin II-Induced Negative Regulation of the Mitogen-Activated Protein Kinases in Cultured Cardiac Myocytes
• 1066 INVOLVEMENT OF ENDOTHELIN -1 IN MECHANICAL STRETCH-INDUCED CARDIAC HYPERTROPHY
• OJ-068 Protective Roles of Heat Shock Transcription Factor-1 in Physiological and Pathological Cardiac Hypertrophy(Cardiac hypertrophy, basic/clinical-2 (M) OJ12,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese C
• Angiotensin II Receptor Blocker Reduces Oxidative Stress and Attenuates Hypoxia-Induced Left Ventricular Remodeling in Apolipoprotein E-Knockout Mice
• FRS-171 Role of Na+-Ca2+ Exchanger in Myocardial Ischemia/Reperfusion Injury : Evaluation Using a Heterozygous Na+-Ca2+ Exchanger Knockout Mouse Model(Coronary Heart Disease : Basic Science (IHD) : FRS20)(Featured Research Session (English))
• Angiotensin-II receptor antagonist combined with β-adrenergic blocker improves left ventricular systolic and diastolic dysfunction in advanced heart failure
• 0802 ANGIOTENSINII IS NOT NECESSARY FOR MECHANICAL STRETCH-INDUCED ACTIVATION OF MITOGEN-ACTIVATED PROTEIN KINASES IN CARDIAC MYOCYTES OF AT1 KNOCKOUT MOUSE
• OJ-145 Anti-inflammatory Effects of MRP8/14 (S100A8/A9) on Experimental Autoimmune Myocarditis by Modulating : Proinflammatory Cytokine Network(OJ25,Myocarditis, Basic/ Clinical (M),Oral Presentation (Japanese),The 73rd Annual Scientific Meeting of The Ja
• PJ-577 Stromal Cell-Derived Factor-1 Is Upregulated in the Infarcted Heart and Mediates Migration of Hematopoietic Stem Cells to the Infarcted Heart(Vasoactive Compound/Growth Factor/Cytokine 2 (H) : PJ97)(Poster Session (Japanese))
• OE-008 G-CSF activates Jak/STAT signaling cascade and increases VEGF expression in cardiac myocytes(Vasoactive Compound/Growth Factor/Cytokine 1 (H) : OE2)(Oral Presentation (English))
• Cardiomyocyte Apoptosis Is a Cause of Heart Failure : A New Mouse Model of Heart Failure
• Functional analysis of CAL, a Cofactor for the Homeobox Transcription Factor CSX / NKX-2.5
• Tbx5 and CsxINkx2-5 associate with each other and bind directly to the ANP promoter in tandem
• Tbx5 associates with Csx/Nkx2-5 and synergistically promotes cardiomyocyte differentiation
• IS037 Both Ca^2+/calmodulin-dependent kinase II and calcineurin play critical roles in cardiac hypertrophy
• IS017 Growth hormone protects cardiac myocytes against H_2O_2-induced apoptosis through activating extracelluar signal-regulated kinase
• Angiotensin II Type 1a Receptor Mediates Doxorubicin-Induced Cardiomyopathy
• Stromal Cell-Derived Factor-1 Mediates Migration of Hematopoletic Stem Cells to the Heart after Myocardial Infarction
• Angiotensin II Type 1a Receptor Plays a Critical Role in Cell Infiltration, Cytokine Production and Angiogenesis in Infarcted Myocardium
• Different gene expressions in volume overload and pressure overload hypertrophy
• Junctophilin-2 plays an important role in isoproterenol-induced cardiomyopathy
• Cardioprotective genes are elevated in physiological hypertrophy
• Targeted lacking of ryanodine receptor type 2 in the heart suppresses pressure overload-induced hypertrophic responses but impairs cardiac functions
• Inhibitory Molecules in Signal Transduction Pathways of Cardiac Hypertrophy
• The disparity of anti-apoptotic mechanism between ischemic preconditioning and pharmacological preconditioning in rabbit heart
• Angiotensin II Induces Hypertrophic Responses through Extracellular Calcium Influx and Activation of CaMKII in Cultured Cardiomyocytes
• -P454- Na/H EXCHANGER MEDIATES STRETCH-INDUCED CARDIACHYPERTROPHY(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
• Overexpression of Heat Shock Factor 1 in the heart protects cardiomyocytes from injury after ischemia/reperfusion
• IS039 Functional Analyses of Three Csx/Nkx2.5 Mutations That Cause Human Congenital Heart Disease
• IS029 Role of TNF-α in oxidative stress-induced apoptosis of cardiac myocytes
• IS016 Extracellular Signal-Regulated Kinases (ERKs) Protect Cardiac Myocytes From Oxidative Stress-Induced Apoptosis
• Molecular and Cellular Mechanisms of Mechanical Stress-Induced Cardiac Hypertrophy
• Continuous Blockade of L-Type Ca^ Channels Suppresses Activation of Calcineurin nad Development of Cardiac Hypertrophy in Spontaneously Hypertensive Rats
• IS046 Rho Family Small G Proteins but not Ras are Important for Mechanical Stress-Induced Hypertrophic Responses in Cardiac Myocytes
• IS044 Role of the Na^+/H^+ exchanger in mechanical stretch-induced cardiac hypertrophy
• P625 MECHANICAL STRESS INDUCES EXPRESSION OF MITOGEN-ACTIVATED PROTEIN KINASE (MAPK) PHOSPHATASE POSSIBLY THROUGH ACTIVATION OF MAPK BUT NOT OF C-JUN N-TERMINAL KINASE IN CARDIAC MYOCYTES.
• 0099 ANGIOTENSIN II INDUCES PROTEIN KINASE C/RAF-1 KINASE/EXTRACELLULAR SIGNAL-REGULATED PROTEIN KINASE ACTIVATION INDEPENDENT OF SRC AND RAS IN CARDIAC MYOCYTES
• -P615- ANATOMICAL AND BIOCHEMICAL REGRESSION OF CARDIACHYPERTROPHY BY A LONG-ACTING CALCIUM ANTAGONIST AMLODIPINE(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
• -0694- MECHANICAL STRESS ACTIVATES PROTEIN KINASE OF PHOSPHORYLATION IN CARDIAC MYOCYTES(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
• -0594- MECHANICAL STRESS INDUCES CARDIAC HYPERTROPHY THROUGH ANGIOTENSIN II-DEPENDENT AND -INDEPENDENT PATHWAYS(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
• -0297- Isolation of Tyrosine Kinases from Adult Murine Heart(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
• -0290- Analysis of the transcription factors involved in differentiation of cardiac myocytes using the in vitro model of embryonic stem cell cardiogenesis.(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
• Infected aortic aneurysm and inflammatory aortic aneurysm-In search of an optimal differential diagnosis

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