The T-type Ca^<2+> Channel Inhibitor Mibefradil Inhibits Voltage-Dependent K^+ Channels in Rabbit Coronary Arterial Smooth Muscle Cells
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概要
- 論文の詳細を見る
- 2012-11-20
著者
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Han Jin
National Research Laboratory For Mitochondrial Signaling Department Of Physiology College Of Medicin
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Hong Da
Institute of Medical Sciences, Department of Physiology, Kangwon National University School of Medicine, Korea
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Yang Dongki
Department of Physiology, Graduate School of Medicine, Gachon University, Korea
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Jung Won-Kyo
Department of Marine Life Science, Chosun University, Korea
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Kim Dae-Joong
Department of Anatomy and Cell Biology, Kangwon National University School of Medicine, Korea
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CHOI Il-Whan
Department of Microbiology, Inje University College of Medicine
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KYOUNG SON
Institute of Medical Sciences, Department of Physiology, Kangwon National University Hospital, Kangwon National University School of Medicine
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HUN NA
Institute of Medical Sciences, Department of Obstetrics and Gynecology, Kangwon National University Hospital, Kangwon National University School of Medicine
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SUN PARK
Institute of Medical Sciences, Department of Physiology, Kangwon National University Hospital, Kangwon National University School of Medicine
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HAN Jin
National Research Laboratory for Mitochondrial Signaling, Department of Physiology, College of Medicine, Cardiovascular and Metabolic Disease Center, Inje University
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JUNG Won-Kyo
Department of Marine Life Science, Chosun University
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HONG Da
Institute of Medical Sciences, Department of Physiology, Kangwon National University Hospital, Kangwon National University School of Medicine
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KIM Dae-Joong
Department of Anatomy and Cell Biology, Kangwon National University Hospital, Kangwon National University School of Medicine
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- The T-type Ca2+ Channel Inhibitor Mibefradil Inhibits Voltage-Dependent K+ Channels in Rabbit Coronary Arterial Smooth Muscle Cells
- Patho-, physiological roles of voltage-dependent K^+ channels in pulmonary arterial smooth muscle cells
- The T-type Ca^ Channel Inhibitor Mibefradil Inhibits Voltage-Dependent K^+ Channels in Rabbit Coronary Arterial Smooth Muscle Cells