Cardioprotection From Ischemia/Reperfusion Injury : Basic and Translational Research

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概要

• 論文の詳細を見る
• 2012-04-25

著者

• MINAMINO Tetsuo

  Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine

• Minamino Tetsuo

  Department Of Cardiovascular Medicine Osaka University Graduate School Of Medicine

関連論文

• Japan Expects Decrements in Both the Incidence and Mortality of Acute Myocardial Infarction in the Modern Era : Likely or Just a Dream?
• 5 Ablation of C/EBP Homologous Protein Attenuates ER-mediated Apoptosis and Cardiac Dysfunction Induced by Pressure Overload(Subcellular Remodeling in the Failing Heart,Symposium 10 (SY-10) (A),The 73rd Annual Scientific Meeting of The Japanese Circulatio
• Inhibition of Cardiac Remodeling by Pravastatin Is Associated with Amelioration of Endoplasmic Reticulum Stress
• Atorvastatin Slows the Progression of Cardiac Remodeling in Mice with Pressure Overload and Inhibits Epidermal Growth Factor Receptor Activation
• OJ-250 A Novel Role of Histamine H2 Receptors in the Pathophysiology of Canine Heart Failure Induced by Sustained Rapid Pacing(Heart failure, basic-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• FRS-092 Chronic Stimulation of Adenosine A2b Receptors Begun after Myocardial Infarction Prevents Cardiac Remodeling in Rats(Heart Failure, Cardiac Remodeling and Cardiomyopathy 2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• PJ-047 The Combined Therapy of Angiotensin Converting Enzyme Inhibitor, Angiotensin II Receptor Blocker and Beta-blockers Submaximally Limits Infarct Size(Acute Myocardial Infarction, Basic 2 (IHD) : PJ8)(Poster Session (Japanese))
• PJ-046 Celiprolol, a Selective β_1-Adrenoceptor Antagonist, Limits Infarct Size via NO-dependent Mechanisms(Acute Myocardial Infarction, Basic 2 (IHD) : PJ8)(Poster Session (Japanese))
• 3 Human Atrial Natriuretic Peptide and Nicorandil as an Adjunct to Reperfusion Therapy for Acute Myocardial Infarction : Lessons from J-WIND Trials(Discovery of Adjunctive Therapies for Acute Myocardial Infarction-From Bench to Bedside-, The 71st Annual S
• The ATP-sensitive Potassium Channel Opener, Nicorandil, Reduces No-reflow Phenomenon and Infarct Size in the Canine Heart
• Polymyxin B-immobilized fiber may improve hemodynamics in patients with severe sepsis and septic shock via the attenuation of nitric oxide
• Nano-size liposome coated with polyethylene glycol can deliver drugs to ischemia/reperfused myocardium(Additional Remarks, Plenary Session 2 (PL-2) (IHD) Myocardial Protection Strategy after Reperfusion Therapy in Acute Coronary Syndrome,Special Program,T
• PJ-092 Activation of Adenosine A1 Receptors Inhibits Aldosterone Synthesis in Adrenal Glands(Metabolism/Biochemistry/Energetics-3 (M) PJ16,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
• Angiotensin II Type 1 Receptor Blocker Prevents Atrial Structural Remodeling in Rats with Hypertension Induced by Chronic Nitric Oxide Inhibition
• OJ-442 Gq-stimulation along with low activity of proteasome induces cardiac apoptosis : Potential mechanism of transition from hypertrophy to failure(Heart Failure, Basic 3 (M) : OJ54)(Oral Presentation (Japanese))
• OJ-324 Antagonism of Aldosterone Receptor Attenuates Severity of Cardiac Dysfunction and Inflammation Induced by Chronic Blockade of NO Synthesis in Rats(Cardiovascular Pharmacology, Basic/Clinical 2 (H) : OJ39)(Oral Presentation (Japanese))
• Canine DNA Array as a Potential Tool for Combining Physiology and Molecular Biology : An Application for the Gene Expression Profile of Regional Ischemic Myocardium
• An Adenosine Analogue, Chloroadenosine, Attenautes Pressure-Overload Myocardial Hypertrophy in Mice
• The New Metalloproteinase Inhibitor Blocks Cardiac Hypertrophy Induced by both Phenylephrine and Angiotensin II
• PJ-195 Activation of Endoplasmic Reticulum-initiated Apoptotic Signaling Mediated by CHOP/GADD153 Contributes to Development of Cardiac Dysfunction in Pressure-overloaded Hearts(Heart failure, basic-6 (M) PJ33,Poster Session (Japanese),The 70th Anniversar
• OJ-320 Granulocyte Colony-Stimulating Factor Mediates Cardioprotection against Ischemia/Reperfusion Injury via Phosphatidylinositol-3-Kinase/Akt Pathway : Evaluation in Vitro Cardiomyocytes and in Vivo Canine Hearts(Acute myocardial infarction, basic-2 (I
• OJ-020 Oxidative Stress-induced Decline of Proteasome Activity Promotes Accumulation of Proapoptotic Proteins and Contributes to Cardiomyocytes Apoptosis in Failing Heart(Heart failure, basic-2 (M) OJ4,Oral Presentation (Japanese),The 70th Anniversary Ann
• The Pathophysiological Role of Endoplasmic Reticulum Stress in the Failing Hearts : Possibility of Brain-Type Natriuretic Peptide as its Marker (Heart Failure, Basic 6 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Neovascularization by Erythropoietin Contributes to the Preservation of Left Ventricular Function after Coronary Artery Ligation in Conscious Beagle Dogs(Angiogenesis 4 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Selective Blockade of Serotonin 5HT2A Receptor Increases Coronary Blood Flow via NO-Dependent Pathway Through 5HT1B Receptor in Hypoperfused Canine Hearts(Coronary Circulation, Basic/Clinical 3 (IHD), The 69th Annual Scientific Meeting of the Japanese Cir
• Nongenomic Deleterious Effects of Aldosterone on Ischemic Hearts via PKC-Dependent Pathway in Dogs(Coronary Circulation, Basic/Clinical 3 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Decreased Proteasome Activity by Oxidative Stress Induces Cardiac Myocytes Apoptosis via p53-mediated Pathway(Heart Failure, Basic 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Cotribution of Endoplasmic Reticulum-Initiated Apoptosis to Progression from Hypertensive to Failing Hearts(Hypertension and Organ Damage, The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• OE-126 The Direct Physiological Impact of C-reactive Protein on Ischemicin Vivo Hearts : Reduced Cardiac Nitric Oxide Production but Saved Metabolization(Coronary Circulation, Basic/Clinical 2 (IHD) : OE15)(Oral Presentation (English))
• Differential Effect on Acute Cardioprotection against Myocardial Infarction by Statins : Role of ECTO-5'-Nucleotidase
• A Selective Estrogen Receptor Modulator, Raloxifene, Inhibits the Cardiac Hypertrophy and Dysfunction due to Aortic Banding in Mice
• Preischemic Protein Kinase-A Activation Triggers Cardioprotection of Ischemic Preconditioning via the Inhibition of Rho-Kinase
• Rationale and Design of a Large-Scale Trial Using Nicorandil as an Adjunct to Percutaneous Coronary Intervention for ST-Segment Elevation Acute Myocardial Infarction : Japan-Working Groups of Acute Myocardial Infarction for the Reduction of Necrotic Damag
• Rationale and Design of a Large-Scale Trial Using Atrial Natriuretic Peptide (ANP) as an Adjunct to Percutaneous Coronary Intervention for ST-Segment Elevation Acute Myocardial Infarction : Japan-Working Groups of Acute Myocardial Infarction for the Reduc
• Rho-kinase Inhibitiors Administered After the Onset of Reperfusion Limit Infarct Size : A Novel Strategy for Adjunctive Therapies Against Myocardial Infarction
• A Role of Transient Preischemic Activation of Protein Kinase-A in Ischemic Preconditioning in Vivo : Involvement of p38 Mitogen-activated Protein Kinase
• Preischemic Protein Kinase-A Activation Evokes Ischemic Preconditioning through Rho-Kinase Inhibition Independent of Protein Kinase-C : A Novel Mechanism
• Amlodipine Increases Coronary Blood Flow and Reduces the Severity of Myocardial Ischemia in Canine Hearts : The Role of NO and Adenosine
• Inhibitory Effects of the CP Compound on Coronary Thromboemboli Caused by an Inhibition of Adenosine Receptors during Coronary Hypoperfusion
• Medical Health Care Checkup Reveals a Novel Risk Factor of Diastolic Heart Dysfunction : Impact of Impaired Glucose Tolerance(Cardiovascular Disease Risk in Diabetes and Glucose Intolerance, The 69th Annual Scientific Meeting of the Japanese Circulation S
• The Long-acting Calcium Channel Blockers Attenuate Cardiac Hypertrophy Induced by Chronic NO Inhibition : The Investigation of the Potential Subcellular Mechanisms
• Gene Expression Profile of Failing Human Hearts In Patients with Dilated Cardiomyopathy Using "Gene Chip" Analysis
• Cellular Mechanisms of Cardioprotection Afforded by Inhibitors of Angiotensin Converting Enzyme in Ischemic Hearts: Role of Bradykinin and Nitric Oxide
• Improvement by 5-Amino-4-Imidazole Carboxamide Riboside of the Contractile Dysfunction That Follows Brief Periods of Ischemia Through Increases in Ecto-5′-Nucleotidase Activity and Adenosine Release in Canine Hearts
• 3 Nano-size Liposomes can Specifically Target Ischemic/Reperfused Myocardium(Latest Development of Nanotechnology Revolutionizes Treatments of Cardiovascular Diseases,Topic 3 (TP-3) (H),The 73rd Annual Scientific Meeting of The Japanese Circulation Societ
• OJ-248 Depression of Proteasome Activities During the Progression of Cardiac Dysfunction in Pressure-overloaded Heart of Mice(Heart failure, basic-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• OJ-298 MEKK1 is Required in Vascular Remodeling after Injury in Carotid Ligation Model(Restenosis after Angioplasty, Basic/Clinical 2 (IHD) : OJ36)(Oral Presentation (Japanese))
• PJ-530 Fasting Serum Phosphate Level : A Potential Marker of Visceral, but not Subcutaneous, Fat Area(Kidney/Renal circulation(07)(H),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
• 2 Statin Prevents Structural and Electrical Atrial Remodeling in Rat Hypertensive Heart Failure Induced by Chronic Inhibition of Nitric Oxide Synthesis(Heart Failure and Arrhythmia, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• Adenosine and Cardioprotection in the Diseased Heart
• Cardioprotection From Ischemia/Reperfusion Injury : Basic and Translational Research

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