Mutations in the Cysteine-Rich Region of the RET Proto-Oncogene in Patients Diagnosed as Having Sporadic Medullary Thyroid Carcinoma
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概要
- 論文の詳細を見る
- 1995-08-01
著者
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Saito S
Univ. Tokushima Tokushima Jpn
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YOSHIMOTO KATSUHIKO
Otsuka Department of Clinical and Molecular Nutrition School of Medicine, The University of Tokushim
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Saito Shiro
The First Department of Internal Medicine, School of Medicine, The University of Tokushima
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Yoshimoto Katsuhiko
Department Of Pharmacology School Of Dentistry The University Of Tokushima
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Kimura T
The First Department Of Internal Medicine School Of Medicine The University Of Tokushima
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KIMURA Takehiko
The First Department of Internal Medicine, School of Medicine, The University of Tokushima
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YOKOGOSHI Yutaka
The First Department of Internal Medicine, School of Medicine, The University of Tokushima
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Saito S
Keio Univ. School Of Medicine Tokyo
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Saito Shiro
The First Department Of Internal Medicine School Of Medicine The University Of Tokushima
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Sasaki Shiro
Department Of Dermatology School Of Medicine The University Of Tokushima
関連論文
- Absence of Mutations at Codon 768 of the RET Proto-Oncogene in Sporadic and Hereditary Pheochromocytomas
- Tumor-Specific Muations in the Tyrosine Kinase Domain of the REN Proto-Oncogene in Pheochromocytomas of Sporadic Type
- Human Pituitary Adenomas Infrequentry Contain Inactivation of Retinoblastoma 1 Gene and Activation of Cyclin Dependent Kinase 4 Gene
- CARDIAC DYSFUNCTION IN PATIENTS WITH CHRONIC PROGRESSIVE EXTERNAL OPHTHALMOPLEGIA
- Infrequent Detectable Somatic Mutations of the RET and Glial Cell Line-derived Neurotrophic Factor (GDNF) Genes in Human Pituitary Adenomas
- Mutations in the Cysteine-Rich Region of the RET Proto-Oncogene in Patients Diagnosed as Having Sporadic Medullary Thyroid Carcinoma
- The Rate of Cell Growth Is Regulated by Purine Biosynthesis via ATP Production and G1 to S Phase Transition
- Immunohistochemical Localization of Activin A and Follistatin in Human Tissues
- Two Different Pituitary Adenomas in a Patient with Multiple Endocrine Neoplasia Type 1 Associated with Growth Hormone-Releasing Hormone-Producing Pancreatic Tumor: Clinical and Genetic Features
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- The Rate of Cell Growth Is Regulated by Purine Biosynthesis via ATP Production and G1 to S Phase Taansition.