スポンサーリンク
Tokyo Institute of Technology Research Laboratory for Nuclear Reactors | 論文
- Association of DNA repair protein NBS1 with telomere maintenance
- The function of NBS1 and histone H2AX in the early process of DNA repair.
- Reversed dose-rate effect of high LET radiation in mutation induction
- Chromosomal instability syndrome of total premature chromatid separation with mosaic variegated aneuploidy is defective in mitotic spindle checkpoint
- Screening of protein interacting with FANCG by Yeast two-hybrid system
- Four novel mutations of the Fanconi anemia group A gene (FAA) in Japanese patients
- Accumulation of Cells at G_2/M Stage by Low Dose-Rate Irradiation Renders the Cell Population More Susceptible to the Subsequent Induction of 6-Thioguanine-Resistant Mutations by ^Cf Fission Neutrons.
- Current Topics in DNA Double-Strand Break Repair
- Functionally important domain analysis of the Nijimegen breakage syndrome gene. NBS1
- Development of a Radio-sensitive Mutation System for the Study of Dose Rate Effects of Space High LET Radiation
- NBS1, the Nijmegen breakage syndrome protein, regulates the localization of DNA repair complex hRAD50/hMRE11/NBS1
- Accelerated telomere shortening by disruption of DNA repair protein NBS1
- Construction of Nbs1 knockout cell line using chicken DT40
- Nijmegen Breakage Syndrome (NBS) gene, NBS1, is involved in telomeric length maintenance.
- CGRP (HUMAN AND ANALOGUE)-INHIBITION OF HUMAN PLATELET AGGREGATION : Myocardial Metabolism, Biochemistry : 53 Annual Scientific Meeting, Japanese Circulation Society
- Induction of dominant lethality and developmental anomalies in offspring of male mice inadiated to varying doses of ^Cf fission neutrons
- Positional cloning of the gene for Nijmegen breakage syndrome
- Genetic Mapping of Nijmegen Breakage Syndrome using Functional Complementation Assays and Homozygosity Mapping
- Haplotype Analysis of Nijmegen Breakage Syndrome and Construction of Physical Map of Candidate Region.
- Complementation Assay between Radiation-Sensitive irs2 Mutant and Ataxia-Telangiectasia Cell