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Division Of Microbiology Kobe University Graduate School Of Medicine | 論文
- Analysis of Neutralizing Antibodies against Hepatitis C Virus in Patients Who Were Treated with Pegylated-Interferon plus Ribavirin
- Autoimmune Thrombocytopenic Purpura during Pegylated Interferon α Treatment for Chronic Hepatitis C
- Histological grading and staging in chronic hepatitis: Its practical correlation
- Full-Length Sequence Analysis of Subacute Sclerosing Panencephalitis (SSPE) Virus, a Mutant of Measles Virus, Isolated from Brain Tissues of a Patient Shortly after Onset of SSPE
- The Phenoxazine Derivative Phx-1 Suppresses IgE-Mediated Degranulation in Rat Basophilic Leukemia RBL-2H3 Cells
- Selective Inhibition of FcεRI-Mediated Mast Cell Activation by a Truncated Variant of Cbl-b Related to the Rat Model of Type 1 Diabetes Mellitus
- Generation of Recombinant Adenovirus Expressing siRNA against the L mRNA of Measles Virus and Subacute Sclerosing Panencephalitis Virus
- An outbreak of fulminant hepatitis B in immunocompromised hemodialysis patients
- A Transgenic Mouse Line with a 58-kb Fragment Deletion in Chromosome 11E1 That Encompasses Part of the Fam20a Gene and Its Upstream Region Shows Growth Disorder
- Hepatitis C virus NS5A protein interacts with 2',5'-oligoadenylate synthetase and inhibits antiviral activity of IFN in an IFN sensitivity-determining region-independent manner
- Inhibition of measles virus and subacute sclerosing panencephalitis virus by RNA interference
- Prediction of Efficient Virological Response to Pegylated Interferon/Ribavirin Combination Therapy by NS5A Sequences of Hepatitis C Virus and Anti-NS5A Antibodies in Pre-Treatment Sera
- Structure and Function of Syk Protein-Tyrosine Kinase^1
- Nonstructural Proteins 4A and 4B of Hepatitis C Virus Transactivate the Interleukin 8 Promoter
- C型慢性肝炎時から肝癌発生まで経過を追えたC型肝炎ウイルスジェノタイプ1bのコア蛋白アミノ酸多様性と肝癌発生との関連性
- Sequence heterogeneity of NS5A and core proteins of hepatitis C virus and virological responses to pegylated-interferon/ribavirin combination therapy
- Inhibition of hepatitis C virus replication through adenosine monophosphate-activated protein kinase-dependent and -independent pathways