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Department of Pharmacology, School of Medicine, University of Occupational and Environmental Health | 論文
- PE-042 Dyslipidemia, Lipid-Rich Atherosclerotic Lesion Formation, and Sudden Death in Mice Lacking All Nitric Oxide Synthases Fed a High-Cholesterol Diet(Atherosclerosis, basic-5, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- OJ-214 Abnormal Glucose Metabolism in Mice Lacking All Nitric Oxide Synthase Isoforms(Genetics/Genetically engineered models/Gene therapy-2(H/M), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- OE-356 Endothelium-Dependent Hyperpolarizations are Totally Dependent on Endothelial Nitric Oxide Synthases System : Lessons from Mice Lacking All Nitric Oxide Synthase Isoforms(Endothelium-1, The 71st Annual Scientific Meeting of the Japanese Circulation
- OE-281 Hyper-Low-Density-Lipoproteinemia in Mice Lacking All Nitric Oxide Synthases(Lipid disorders-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- Propofol Inhibits Muscarinic Acetylcholine Receptor-Mediated Signal Transduction in Xenopus Oocytes Expressing the Rat M1 Receptor
- EFFECTS OF CLASS I ANTIARRHYTHMIC DRUGS ON ADRENOCEPTORS IN CULTURED RAT CARDIOCYTES
- Long-Term Treatment With Imidapril but Not With Nifedipine Enhances Plasma NOx Concentration in Patients With Essential Hypertension
- Treatment with imidapril but not with nifedipine increases plasma NOx concentrations in patients with essential hypertension
- Long-Term Blockade of Angiotensin-Converting Enzyme Ameliorates Dobutamine-Induced Myocardial Ischemia in Patients with Coronary Artery Disease
- Long-Term Treatment With Perindopril Ameliorates Dobutamine-Induced Myocardial Ischemia in Patients With Coronary Artery Disease
- Direct Inhibition of N-Methyl-D-Aspartate (NMDA)-Receptor Function by Antiglaucomatous β-Antagonists
- PE-433 Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthase Isoforms(Diabetes/Obesity/Metabolic syndrome-4 (H) PE73,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- OE-199 Genetic Disruption of All Nitric Oxide Synthase Isoforms Markedly Reduces Survival in Mice(Endothelium-1 (H) OE34,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- OE-133 Vasculoprotective Role of the Nitric Oxide Synthases System against Vascular Lesion Formation in Mice in Vivo(Peripheral circulation-1 (H) OE23,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation S
- OE-063 Spontaneous Development of Systemic Arteriosclerosis in Mice Lacking All Nitric Oxide Synthase Isoforms(Atherosclerosis, basic-1 (H) OE11,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society
- Inhibition by Selenium Compounds of Catecholamine Secretion Due to Inhibition of Ca^ Influx in Cultured Bovine Adrenal Chromaffin Cells
- Effect of cAMP-Dependent Protein Kinase on Catecholamine Secretion from Bovine Adrenal Chromaffin Cells
- FRS-084 Spontaneous Myocardial Infarction in Mice Lacking All Nitric Oxide Synthase Isoforms(Molecular Biology in Cardiovascular Diseases (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- FRS-021 Long-Term Treatment with an Angiotensin II Type 1 Receptor Blocker Ameliorates Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthases(Diabetes/Obesity (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- Development of Genetically Engineered Mice Lacking All Three Nitric Oxide Synthases
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