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山口大学 医学系研究科 | 論文
- PJ-367 Ser136 Phosporylation of Bad by PKC Activation is a New Therapeutic Strategy for Improving the Muscle-derived Stem Cell Survival(PJ062,Regeneration (Basic, Clinical) (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese C
- Regression of abdominal aortic aneurysms by pharmacological inhibition of c-Jun N-terminal kinase
- FRS-058 A Novel Stem Cell Source for Cardiomyocyte Regeneration(Regeneration Research in Cardiovascular Medicine (M) FRS12,Featured Research Session,The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-430 Domain2114-2149 within Ryanodine Receptor (RyR) Plays a Key Role for Stabilization of RyR2 in Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT)(PJ073,Arrhythmia, Others (Basic) 1 (A),Poster Session (Japanese),The 73rd Annual Scientific M
- PJ-073 Protein Phosphatase 1 Beta is Most Significant Subunit in the Longitudinal Sarcoplasmic Reticulum and Controles Phospholamban Phosphorylation in Cardiomyocytes(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd
- PJ-071 In Vivo Knockdown of Protein Phosphatase 1β Augments Cardiac Contracitility in Mice(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
- PJ-068 Abnormally Tight Domain-Domain Interaction at Mutation Site could be a Primary Cause of Catecholaminergic Polymorphic Ventricular Tachycardia(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific
- 5 Decreased affinity of calmodulin binding to RyR2 may cause leaky channel in CPVT-associated mutation(Young Investigator's Award for International Students Finalists Lecture (YIA International),The 73rd Annual Scientific Meeting of The Japanese Circulati
- 3 Correction of RyR2 Defects as a New Therapeutic Strategy against Heart Failure(Novel Therapeutic Strategies against Heart Failure,KSC-JCS Joint Symposium (KSC-JCS Joint Symposium) (I),The 73rd Annual Scientific Meeting of The Japanese Circulation Societ
- Defective Ca^ Cycling as a Key Pathogenic Mechanism of Heart Failure(Molecular Mechanisms)
- FRS-018 Marked Increase in Spontaneous Ca2+ Sparks In Response To β-stimulation As A Critical Cause Of Catecholaminergic Polymorphic Ventricular Tachycardia(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scien
- FRS-017 Enhanced Sensitivity of the Cardiac Ryanodine Receptor to Activation by Luminal Ca^ as a Primary Cause of CPVT(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circ
- 4 Phospho-regulation of Sarcoplasmic Reticulum Microdomain in Heart Failure(Symposium 7 (SY-07) (M) Molecular Mechanism and Novel Therapeutic Target in Heart Failure,Special Program,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-765 Scavenging Free Radicals by Low-dose Carvedilol Prevents Redox-dependent Ca^ Leak via Stabilization of Ryanodine Receptor in Heart Failure(Development and differentiation-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society
- OJ-249 Interruption of Specific Domain-domain Interaction within Ryanodine Receptor Mediates Stabilization of Ryanodine Receptor in Failing Hearts(Heart failure, basic-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- AT_1 Receptor Antagonist Restores Cardiac Ryanodine Receptor Function, Rendering Isoproterenol-Induced Failing Heart Less Susceptible to Ca^-Leak Induced by Oxidative Stress(Experimental Investigation)
- OJ-295 Effect of Tautomycin, a Chemical Inhibitor of Protein Phosphatase 1, on Cellular Contractility and Calcium Transient in Adult Rat Cardiomyocytes(Cardiac function, basic/clinical-2 (M) OJ50,Oral Presentation (Japanese),The 70th Anniversary Annual Sc
- Tongue Muscle-Derived Stem Cells Express Connexin 43 and Improve Cardiac Remodeling and Survival After Myocardial Infarction in Mice
- PE-164 Defective Inter-domain Interaction may Cause Spontaneous Ca^ Release via Reduced Affinity of Calmodulin Binding to RyR2 in Failing Hearts(PE028,Heart Failure, Basic (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese
- Defective Regulation of Inter-domain Interactions within the Ryanodine Receptor Plays a Key Role in the Pathogenesis of Heart Failure (Excitation-contraction Coupling/Ion Channel 2 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Societ
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