Makita Naomasa | Department Of Cadiovascular Medicine Hokkaido University Graduate School Fo Medicine

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著作論文

State-Dependent Blocking Actions of Azimilide Dihydrochloride (NE-10064) on Human Cardiac Na^+ Channels
L-Cysteine Prevents Oxidation-Induced Block of the Cardiac Na^+ Channel Via Interaction With Heart-Specific Cysteinyl Residues in the P-Loop Region
OE-389 Clinical Phenotype of Japanese LQT1 Form of Congenital Long QT Syndrome by Location and Coding Type From Japanese Multicenter Registry(Arrhythmia, diagnosis/Pathophysiology/EPS(02)(A),Oral Presentation(English),The 72nd Annual Scientific Meeting of
Absence of a Trafficking Defect in R1232W/T1620M, a Double SCN5A Mutant Responsible for Brugada Syndrome
1 Molecular Basis for Low Penetrance in the Long QT Syndrome(Meet the Expert 7 (ME7) (A) : Issues on Sudden Cardiac Death)(Special Program)
OE-133 Novel intrinsic mechanism of the enhanced rate-dependant QT Shortening in R1623Q mutant of the LQT3 syndrome(Arrhythmia, Basic 4 (A) : OE16)(Oral Presentation (English))
OJ-009 A Common KCNE1 Polymorphism, D85N, is a Genetic Modifier of Long QT Syndrome(Arrhythmia, diagnosis/ Pathophysiology/ EPS(03)(A),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
A single mutation of cardiac sodium channel gene confers phenotypes of both Brugada and long QT syndromes
Ion channel mutations and polymorphisms in Japanese patients with acquired long QT syndrome
PE-103 Clinical Phenotype of Japanese LQT3 Form of Congenital Long QT Syndrome by Location of Mutation from Japanese Multicenter Registry(PE018,Arrhythmia, Others (Clinical/Pathophysiology) (A),Poster Session (English),The 73rd Annual Scientific Meeting o
FRS-117 Clinical and Electrocardiographic Characteristics of Japanese Probands with Brugada Syndrome with an SCN5A Mutation from Japanese Multicenter Registry(FRS24,Novel Mechanisms of Ventricular Arrhythmia (A),Featured Research Session (English),The 73r
PJ-208 Impact of Frameshift Mutation and Nonsense-mediated mRNA Decay on Clinical Phenotype in LQT2 Form of Congenital Long QT Syndrome(Arrhythmia,diagnosis/Pathophysiology/EPS(13)(A),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japa
Congenital atrial standstill associated with coinheritance of a novel SCN5A mutation and connexin 40 polymorphisms
FRS-119 Familial Isolated Cardiac Conduction Defect Associated with a Mutation in the Connexin40 Gene GJA5(FRS24,Novel Mechanisms of Ventricular Arrhythmia (A),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulat
An SCN5A Mutation Associated With Idiopathic Ventricular Fibrillation Alters The Na^+ Channel Pore Structure And Responses To Antiarrhythmic Drugs
PJ-190 A KCNE1 Single Nucleotide Polymorphism, D85N, Serves as a Modifier in Forme-fruste Type of Long QT syndrome(Genetics/Genetically engineered models/Gene therapy-3 (M) PJ32,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of t
Novel SCN5A mutation (Q55X) associated with age-dependent expression of Brugada syndrome presenting as neurally mediated syncope.
Double SCN5A mutation underlying asymptomatic Brugada syndrome
Single Nucleotide Polymorphisms of Cardiac Na^+ Channel Gene SCN5A : Allelic Prevalence in Brugada Syndrome and Their Biophysical Properties
High-risk for Bradyarrhythmic Complications in Patients with Brugada Syndrome Caused by SCN5A Gene Mutations(Arrhythmia, Basic (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
5 Overlap Between LQT3 and Brugada Syndrome : Clinical Features in a Common Mutation and Underlying Biophysical Mechanisms(Plenary Session 4 (PL-4) (A) New Scientific Approaches to QT Prolongation,Special Program,The 72nd Annual Scientific Meeting of the
NORADRENALINE-INDUCED ALTERATIONS IN ADENYLATE CYCLASE SYSTEM OF RAT HEART MEMBRANES : Cardiovascular Drugs : 53 Annual Scientific Meeting, Japanese Circulation Society
Genetic Polymorphisms and Arrhythmia Susceptibility(Genetics of Arrhythmias)
Molecular Basis for the Phenotypic Variability of Cardiac Sodium Channelopathies(Ion Channels and Cardiac Arrhythmias : Current Understanding and Future, The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PE-307 Left Ventricular Noncompaction Associated With Mutations in Cardiac Na Channel Gene SCN5A(Cardiomyopathy, basic/clinical-4, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-085 Genetic Factors Conferring Congenital Sick Sinus Syndrome in SCN5A Mutation Carriers(Arrhythmia, basic-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
ALTERATIONS OF PHOSPHOINOSITIDE-SPECIFIC PHOSPHOLIPASE C AND PROTEIN KINASE C ACTIVITIES IN THE MYOCARDIUM OF SPONTANEOUSLY HYPERTENSIVE RATS (SHR) : Cardiomyopathy, Myocarditis : 53 Annual Scientific Meeting, Japanese Circulation Society
-P163- PARTIAL PURIFICATION AND CHARACTERIZATION OF PHOSPHATIDYLINOSITOL-SPECIFIC PHOSPHOLIPASE C IN RAT MYOCARDIUM
1 Genotype-Phenotypes Relations of SCN5A-Positive Brugada Syndrome(Brugada Syndrome:From Cell to Bedside,Plenary Session 2 (PL2) (A),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Aborted Sudden Cardiac Death Associated with Short QT Syndrome
Enhanced sodium channel intermediate inactivation in Brugada syndrome
Molecular and Biophysical Bases of Brugada Syndrome
-0532-ALTERATIONS OF MEMBRANE PHOSPHOINOSITIDE METABOLISM IN THE HEART OF CARDIOMYOPATHIC SYRIAN HAMSTER BIO 14.6 (BIO)
-0229-ALTERATIONS OF PHOSPHOLIPASE C (PLC) ISOZYME PATTERN IN THE AORTA OF SPONTANEOUSLY HYPERTENSIVE RATS (SHR)
Guidelines for Risks and Prevention of Sudden Cardiac Death (JCS 2010) : Digest Version
Novel SCN3B Mutation Associated With Brugada Syndrome Affects Intracellular Trafficking and Function of Nav1.5
Novel SCN3B Mutation Associated Witt Brugada Syndrome Affects Intracellular Trafficking and Function of Nav1.5

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