MAKITA Naomasa | Department of Cardiovascular Medicine, Hokkaido University Graduate School of Medicine

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著作論文

Phenotypic Overlap of Cardiac Sodium Channelopathies : Individual-Specific or Mutation-Specific?
State-Dependent Blocking Actions of Azimilide Dihydrochloride (NE-10064) on Human Cardiac Na^+ Channels
L-Cysteine Prevents Oxidation-Induced Block of the Cardiac Na^+ Channel Via Interaction With Heart-Specific Cysteinyl Residues in the P-Loop Region
Absence of a Trafficking Defect in R1232W/T1620M, a Double SCN5A Mutant Responsible for Brugada Syndrome
1 Molecular Basis for Low Penetrance in the Long QT Syndrome(Meet the Expert 7 (ME7) (A) : Issues on Sudden Cardiac Death)(Special Program)
OE-133 Novel intrinsic mechanism of the enhanced rate-dependant QT Shortening in R1623Q mutant of the LQT3 syndrome(Arrhythmia, Basic 4 (A) : OE16)(Oral Presentation (English))
OJ-009 A Common KCNE1 Polymorphism, D85N, is a Genetic Modifier of Long QT Syndrome(Arrhythmia, diagnosis/ Pathophysiology/ EPS(03)(A),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
A single mutation of cardiac sodium channel gene confers phenotypes of both Brugada and long QT syndromes
Ion channel mutations and polymorphisms in Japanese patients with acquired long QT syndrome

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