Kojima Tetsuhito | Department Of Internal Medicine Nagoya University School Of Medicine

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著作論文

Activated protein C resistance in the Japanese population due to homozygosity for the factor V R2 haplotype
Stress-induced PAI-1 expression is suppressed by pitavastatin in vivo
Definite diagnosis in Japanese patients with protein C deficiency by identification of causative PROC mutations
Mutational analysis of SOS1 gene in acute myeloid leukemia
Identification of amino acid residues responsible for von Willebrand factor binding to sulfatide by charged-to-alanine-scanning mutagenesis
Mutated RAS Induced PLD1 Gene Expression through Increased Sp1 Trascription Factor
Molecular Defects Associated with Antithrombin Deficiency and Dilated Cardiomyopathy in a Japanese Patient
Recurrent Mutations of Factor XI Gene in Japanese
Recurrent Intramural Hematoma of the Small Intestine in a Severe Hemophilia A Patient with a High Titer of Factor VIII Inhibitor : A Case Report and Review of the Literature
High Titer of ADAMTS13 Inhibitor Associated with Thrombotic Microangiopathy of the Gut and Skeletal Muscle after Allogeneic Hematopoietic Stem Cell Transplantation
Severe hemophilia A in a Japanese female caused by an F8-intron 22 inversion associated with skewed X chromosome inactivation
Classic Polyarteritis Nodosa Presenting Rare Clinical Manifestations in a Patient with Hemophilia A
Identification of the regulatory elements of the human von Willebrand factor for binding to platelet GPIb. Importance of structural integrity of the regions flanked by the CYS1272-CYS1458 disulfide bond
Virus-Associated Hemophagocytic Syndrome Caused by Pandemic Swine-Origin Influenza A (H1N1) in a Patient After Unrelated Bone Marrow Transplantation
リュードカン : その分子構造と機能
Current Venous Thromboembolism Management and Outcomes in Japan:– Nationwide The Japan Venous Thromboembolism Treatment Registry Observational Study –
Transcriptional regulation of neutral sphingomyelinase 2 in all-trans retinoic acid-treated human breast cancer cell line, MCF-7
Role of down-regulated neutral ceramidase during all-trans retinoic acid-induced neuronal differentiation in SH-SY5Y neuroblastoma cells

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