A study on lymphokine activated killer(LAK) cell activity in inflammatory bowel disease(IBD).
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The present study was designed to investigate if a difference exists in the capacity to generate lymphokine activated killer (LAK) cells, which can be generated by in vitro exposure lymphocytes to interleukin-2 (IL-2), between inflammatory bowel disease (IBD) and normal controls. <BR>The results are decribed as below. <BR>1) In active Crohn's disease patients, LAK cell activity was slightly lower than that in normal controls, but there was no significant difference. <BR>2) The sera of IBD patients showed no significant effect on the induction of LAK cell activity as compared with normal lymphocytes. <BR>3) Physiological concentration of dexamathasone (DEX) significantly reduced LAK cell activity during the LAK inducer phase. LAK cell activity could be correlated to the concentration of DEX during the LAK effector phase. <BR>4) Salicylazosulfapyridine and its metabolite (0.1 mM) showed no significant effect on the induction of LAK cell activity. <BR>In conclusion, LAK cell activity by exogenouse IL-2 was not impaired in IBD patients.
- 日本大腸肛門病学会の論文
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