High-Output Heart Failure in Mitochondrial Myopathy: A Fulminant Form with Severe Lactic Acidosis and Rhabdomyolysis.
スポンサーリンク
概要
- 論文の詳細を見る
A 17-year-old male with mitochondrial myopathy was transferred to our hospital because of respiratory distress and a rapidly deteriorating level of consciousness. He had a markedly increased cardiac output with low systemic vascular resistance and decreased arteriovenous oxygen difference. Severe lactic acidosis, rhabdomyolysis and acute renal failure were also present. He was treated in the intensive care unit with continuous hemofiltration and hemodialysis. This case demonstrates that cardiac failure in mitochondrial encephalomyopathy may be caused by myocardial abnormalities as well as by reduced systemic vascular resistance secondary to disturbed oxidative metabolism.(Internal Medicine 32: 798-801, 1993)
- 社団法人 日本内科学会の論文
著者
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Higuma Norio
The Department Of Cardiology Niigata City General Hospital
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Oda Hirotaka
The Department Of Cardiology Niigata City General Hospital
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Miida Tsutomu
The Department Of Cardiology Niigata City General Hospital
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HIROSE Yasuo
the Critical Care Medical Center, Niigata City General Hospital
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TOEDA Tetsuro
the Department of Cardiology, Niigata City General Hospital
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YOSHIDA Kazukiyo
the Department of Nephrology, Niigata City General Hospital
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KIMURA Hideki
the Department of Nephrology, Niigata City General Hospital
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- High-Output Heart Failure in Mitochondrial Myopathy: A Fulminant Form with Severe Lactic Acidosis and Rhabdomyolysis.