Marked Increase in Fundic Mucosal Histidine Decarboxylase Activity in a Patient with H+,K+-ATPase Antibody-Positive Autoimmune Gastritis.
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概要
- 論文の詳細を見る
A 63-year-old woman was diagnosed as autoimmune gastritis by the presence of serum antibody against α-subunit of gastric H+, K+-ATPase. The patient did not have pernicious anemia, but showed achlorhydria, marked hypergastrinemia, enterochromaffin-like cell hyperplasia and an extremely high histidine decarboxylase activity in the gastric fundic mucosa. Intragastric acidification by infusion of hydrochloric acid via a nasogastric tube induced a transient reduction of serum gastrin level and fundic mucosal histidine decarboxylase activity. A marked increase in fundic mucosal histidine decarboxylase activity as well as hypergastrinemia appears to be the pathophysiologic response to achlorhydria caused by autoimmunity against gastric H+, K+-ATPase.(Internal Medicine 32: 602-606, 1993)
- 社団法人 日本内科学会の論文
著者
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Matsuzawa Yuji
The Second Department Of Internal Medicine Osaka University Medical School
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FUTAI Masamitsu
the Department of Organic Chemistry and Biochemistry, Institute of Scientific and Industrial Research, Osaka University
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MIYAXAKI Yoshiji
the Second Department of Internal Medicine, Osaka University Medical School
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SHINOMURA Yasuhisa
the Second Department of Internal Medicine, Osaka University Medical School
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MURAYAMA Yoko
the Second Department of Internal Medicine, Osaka University Medical School
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IMAMURA Ikuo
the Department of Pharmacology II, Osaka University Medical School
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FUKUI Hiroyuki
the Department of Pharmacology II, Osaka University Medical School
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MAEDA Masatomo
the Department of Organic Chemistry and Biochemistry, Institute of Scientific and Industrial Research, Osaka University
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