Differential Regulation of Extracellular Signal-Related Kinase Phosphorylation by Vitamin D3 Analogs

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概要

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• We examined the effects of vitamin D3 on extracellular signal-related kinase (ERK). 1,25-(OH)2D3, a form active in inducing transcription, caused rapid and transient ERK activation. 24R,25-(OH)2D3, an inactive form, also activated ERK. In contrast, (22R)-22-methyl-20-epi-1,25-(OH)2D3, a synthetic agonist of 1,25-(OH)2D3, was not effective. These data provide evidence of selective roles of vitamin D3 in nongenomic and genomic actions.

• 社団法人 日本農芸化学会の論文

著者

• YAMAUCHI Jun

  Nutritional Epidemiology Program, National Institute of Health and Nutrition

• ISHIMI Yoshiko

  Nutritional Epidemiology Program, National Institute of Health and Nutrition

• Yamauchi Jun

  Nutritional Epidemiology Program Bio-index Project National Institute Of Health And Nutrition

• INOUE Erina

  Nutritional Epidemiology Program, Bio-Index Project, National Institute of Health and Nutrition

関連論文

• Effect of Low Dose Vitamin K2 (MK-4) Supplementation on Bio-Indices in Postmenopausal Japanese Women
• Differential Regulation of Extracellular Signal-Related Kinase Phosphorylation by Vitamin D_3 Analogs
• Differential Regulation of Extracellular Signal-Related Kinase Phosphorylation by Vitamin D3 Analogs

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