Over-activation of cAMP signaling increases vulnerability to hydrogen peroxide via actin rearrangement, which is inhibited by prostaglandin E2 in a mouse granulosa cell line.
スポンサーリンク
概要
著者
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Aoyama Yasunori
Department Of Morphophysiology Faculty Of Pharmaceutical Science Josai International University
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AOYAMA Yasuhiro
Department of Synthetic Chemistry and Biological Chemistry, Graduate School of Engineering, Kyoto Un
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Asakai Rei
Department of Endocrinology, Tokyo Medical and Dental University
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Koushi Masami
Molecular Pathophysiology, Department of Moleculo-genetic Sciences, Division of Biomedical Laborator
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Kamei Yoshiko
Department of Morphophysiology, Faculty of Pharmaceutical Sciences, Josai International University
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Takizawa Toichiro
Molecular Pathophysiology, Department of Moleculo-genetic Sciences, Division of Biomedical Laborator
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Asakai Rei
Department Of Morphophysiology Faculty Of Pharmaceutical Science Josai International University
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Asakai Rei
Department Of Endocrinology Tokyo Medical And Dental University
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Koushi Masami
Molecular Pathophysiology Department Of Moleculo-genetic Sciences Division Of Biomedical Laboratory
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Kamei Yoshiko
Department Of Morphophysiology Faculty Of Pharmaceutical Science Josai International University
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Takizawa Toichiro
Molecular Pathophysiology Department Of Moleculo-genetic Sciences Division Of Biomedical Laboratory
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Aoyama Yasuhiro
Department Of Chemistry Nagaoka University Of Technology
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- Over-activation of cAMP signaling increases vulnerability to hydrogen peroxide via actin rearrangement, which is inhibited by prostaglandin E2 in a mouse granulosa cell line.
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