OJ-172 Inactivation of Bach1 Promotes Macrophage-phagocytosis through the Induction of Heme Oxygenase-1 (HO-1) and Monocyte Chemoattachment Protein-1 (MCP-1)(Infection/Inflammation/Immunity 1 (H) : OJ20)(Oral Presentation (Japanese))
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概要
- 論文の詳細を見る
- 社団法人日本循環器学会の論文
- 2004-03-01
著者
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Yano Yoko
Department Of Clinical Laboratory Medicine Hiroshima University Graduate School Of Biomedical Scienc
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Omura Shinji
Departments Of Biomedical Chemistry Hiroshima University Graduate School Of Biomedical Sciences
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Omura Shinji
Department Of Biomedical Chemistry Graduate School Of Biomedical Sciences Hiroshima University
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Igarashi Kazuhiko
Departments Of Biomedical Chemistry Hiroshima University Graduate School Of Biomedical Sciences
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Igarashi Kazuhiko
Department Of Biochemistry Hiroshima University School Of Medicine
関連論文
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- Modulation of Target Tissue Response to Angiotensin II and Sodium Sensitivity in Japanese Patients with Essential Hypertension
- Induction of Heme Oxygenase-1 Inhibits Monocyte Chemoattractant Protein-1 mRNA Expression in U937 Cells
- Insulin Resistance, Insulin Secretion, and Cardiovascular Mortality : Data from Hawaii-Los Angeles-Hiroshima Study
- Glucose Tolerance and Cardiovascular Mortality : Comparison of 1997 American Diabetes Association and World Health Organization Criteria
- Cardioprotective Role of AT2 Receptor in Post-Infarction Left Ventricular Remodeling
- Bach1 Deficiency Ameliorates Hepatic Injury in a Mouse Model
- Beneficial Effect of T-Type Calcium Channel Blockers on Endothelial Function in Patients with Essential Hypertension
- Genetic Ablation of Bach 1, a Critical Transcriptional Repressor of Heme Oxygenase-1, Prevents Left Ventricular Remodeling after Myocardial Infarction in Mice(Old Myocardial Infarction/Remodeling 3 (IHD), The 69th Annual Scientific Meeting of the Japanese
- Relationship between HO-1 Upregulation Capacity in PBMC and Severity of Coronary Hear Disease(Angina Pectoris, Basic/Clinical 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- FRS-163 Genetic ablation of Bach1, a critical transcriptional repressor of heme oxygenase-1, leads to myocardial protection against ischemia/reperfusion injury in mice(Coronary Heart Disease : Basic Science (IHD) : FRS20)(Featured Research Session (Englis
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- PE-175 Genetic Ablation of Bach1, a Transcriptional Repressor of Heme Oxygenase-1 Gene, Leads to Myocardial Protection against Pressure Overload in Mice(Cardiac hypertrophy, basic/clinical-3 (M) PE30,Poster Session (English),The 70th Anniversary Annual Sc
- BACH1 DEFCIENCY AMELIORATES DEXTRAN SULPHATE SODIUM-INDUCED COLITIS IN MICE
- Effects of Genetic Ablation of Bach1 upon Smooth Muscle Cell Proliferation and Atherosclerosis after Cuff Injury(Molecular Biology, Vascular 1 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Heme-Regulated Transcription Factor Bach1
- Differential activation of mitogen-activated protein kinases and glial cells in the trigeminal sensory nuclear complex following lingual nerve injury
- Regulation of the plasma cell transcription factor Blimp-1 gene by Bach2 and Bcl6
- Architecture and Dynamics of the Transcription Factor Network that Regulates B-to-Plasma Cell Differentiation
- PE-317 Bach1 Is a Key-repressor of Heme Oxygenase-1 (HO-1) and Regulates Cell Proliferation(Atherosclerosis, Basic 4 (IHD) : PE54)(Poster Session (English))
- OJ-172 Inactivation of Bach1 Promotes Macrophage-phagocytosis through the Induction of Heme Oxygenase-1 (HO-1) and Monocyte Chemoattachment Protein-1 (MCP-1)(Infection/Inflammation/Immunity 1 (H) : OJ20)(Oral Presentation (Japanese))
- Dynamic Cytoplasmic Anchoring of the Transcription Factor Bach1 by Intracellular Hyaluronic Acid Binding Protein IHABP
- Co-repressor SMRT and Class II Histone Deacetylases Promote Bach2 Nuclear Retention and Formation of Nuclear Foci that are Responsible for Local Transcriptional Repression
- Expression of the Oxidative Stress-Regulated Transcription Factor Bach2 in Differentiating Neuronal Cells
- Chromatin and Cancer: The Eleventh International Symposium of the Hiroshima Cancer Seminar, November 2001
- Gas-particle heat transfer in a packed fluidized bed.