Tyrosine Kinase of the Insulin-like Growth Factor I Receptor Is Dispensable for Heat Shock Activation of MAPK

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Radioresistant signaling mechanism induced by Insulin-like growth factor I receptor (IGF-IR) in 3Y1 cells lacking Insulin receptor substarate-1 (IRS1)
Lack of Relationship between Expression of the Insulin-like Growth Factor I Receptor and Radiosensitivity inAtaxia Telangiectasia (A-T) Cells
Effect of the Insulin-like growth factor I receptor (IGF-IR) on radiosensitivity of human glioblastoma cells grown in multicellular spheroids.
Tyrosine Kinase of the Insulin-like Growth Factor I Receptor Is Dispensable for Heat Shock Activation of MAPK
Ionizing Radiation Activates Caspase-8/FLICE in Mouse Embryo Fibroblasts with A Targeted Disruption of The Insulin-like Growth Factor I Receptor Gene
Biological Responses to Ionizing Radiation in Mouse Embryo Fibroblasts with A Null Mutation of the DNA Polymerase β Gene
Redundancy of Radioresistant Signaling Pathways Originating from Insulin-like Growth Factor I Receptor
The PI3 kinase/Akt Survival Pathway Is Not Required for IGF-I Receptor-mediated Clonogenic Radioresistance
Base Excision Repair of MMS-Induced Base Damages in Pol β Deficient Mouse Cells
140 Inhibition of the MEK/ERK Pathway Induces Radioresistance in Rat 3Y1 Cells(Redox regulation, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
The Phosphatidylinositol-3 kinase Pathway Is Not Essential for Insulin-like Growth Factor I Receptor-mediated Clonogenic Radioresistance.

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