Redundancy of Radioresistant Signaling Pathways Originating from Insulin-like Growth Factor I Receptor
スポンサーリンク
概要
著者
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Yu Dong
Mol. Diag. Ther. Grad. Sch. Tokyo Med. Dent. Univ.
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MIURA Masahiko
Mol. Diag. Ther. Tokyo Med. and Dent. Univ.
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Miura Masahiko
Mol. Diag. Ther. Grad. Sch. Tokyo Med. Dent. Univ.
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SHIBUYA Hitoshi
Diag. Radiol. Oncol. Grad. Sch. Tokyo Med. Dent. Univ.
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Miura Masahiko
Mol. Diag. Grad. Sch. Tokyo Med. And Dent. Univ.
関連論文
- Radioresistant signaling mechanism induced by Insulin-like growth factor I receptor (IGF-IR) in 3Y1 cells lacking Insulin receptor substarate-1 (IRS1)
- Lack of Relationship between Expression of the Insulin-like Growth Factor I Receptor and Radiosensitivity inAtaxia Telangiectasia (A-T) Cells
- Effect of the Insulin-like growth factor I receptor (IGF-IR) on radiosensitivity of human glioblastoma cells grown in multicellular spheroids.
- Tyrosine Kinase of the Insulin-like Growth Factor I Receptor Is Dispensable for Heat Shock Activation of MAPK
- Redundancy of Radioresistant Signaling Pathways Originating from Insulin-like Growth Factor I Receptor
- The PI3 kinase/Akt Survival Pathway Is Not Required for IGF-I Receptor-mediated Clonogenic Radioresistance
- 140 Inhibition of the MEK/ERK Pathway Induces Radioresistance in Rat 3Y1 Cells(Redox regulation, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- 145 Roles of the Insulin-like Growth Factor I Receptor C-terminus in Clonogenic Radioresistance(Redox regulation, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)