Are defective, HIV protease-deficient particles the real culprit in AIDS?A specific type of defective particle produced after @@S ' persistent infection of lymphoid cells may be the trigger in @@S ' pathogenesis.

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A specific T-cell subset with CD4+/CD38- markers derived from HIV-1 cariers induses apoptosis in healthy donor-derived T-lymphocytes.
Dependence of host cell cycle for activation of human immunodeficiency virus type 1 gene expression from latency
Generation of endogenous tumour necrosis factor-α in MOLT-4 cells during the acute replication phase of subsequent latent infection.
E Detection of Theileria sergenti schizonts in bovine lymph node.
Inhibitory effect of heparin on red blood cell invasion by Theileria sergenti merozoites.
Superoxide generation by monocytes following infection with human cytomegalovirus.
Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25+ killer cells in a small subset, leading to induction of apoptosis in bystander cells.
Inhibition of HIV-1 replication by targeting the Rev protein.
Demonstration of Borna disease virus (BDV) in specific regions of the brain from horses positive for serum antibodies to BDV but negative for BDV RNA in the blood and internal organs.
High prevalence of Borna disease virus infection in healthy sheep in Japan.
Production of doughnut-shaped, protease-defective particles from a T cell clone carrying a provirus with specific mutation in the env, pol, vpr,and nef genes.
E Decoy approach using RNA-DNA chimera oligonucleotides to inhibit the regulatory function of human immunodeficiency virus type 1 Rev protein
Protease-defective, gp120-containing human immunodeficiency virus type 1 particles induce apoptosis more efficiently than does wild-type virus or recombinant gp120 protein in healthy donor-derived peripheral blood T cells.
Possible Correlation between Borna Disease Virus Infection and Japanese Patients with Chronic Fatigue Syndrom
Demonstration of Borna disease virus RNA in peripheral blood mononuclear cells from healthy horses in Japan.
Prevalence of Borna disease virus RNA in peripheral blood mononuclear cells from blood donors.
Viral activation from latency during retrodifferentiation of U937 cells exposed to phorbol ester followed by infection with human immunodeficiency virus type 1.
High susceptibility of U937-derived subclone to infection with human immunodeficiency virus type 1 is correlated with virus-induced celldifferentiation and superoxide generation.
Stimulation of human immunodeficiency virus type 1 infected cells with superoxide enhances the chemotactic motile response of CD4+ human cells: implication for virus transmission by cell-to-cell interaction.
Intracellular localization and DNA-binding activity of a class of viral early phosphoproteins in human fibroblasts infected with @@S ' human cytomegalovirus (Towne strain)
Amplification of a Full-Length Borna Disease Virus(BDV) cDNA from Total RNA of Cells Persistently Infected with BDV
Superinfection of a defective human immunodeficiency virus type 1 provirus-carrying T cell clone with vif or vpu mutants gives cytopathic virus particles by homologous recombination.
Naturally occurring accessory gene mutations lead to persistent human immunodeficiency virus type 1 infection of CD4-positive T cells.
Nonsense mutations in the vpr gene of HIV-1 during in vitro virus passage and in HIV-1 carrier-derived peripheral blood mononuclear @@S ' cells.
Genetic and functional variabilities in the human immuno @@S deficiency virus tyre 1 isolated from the same blood samples @@S ' with and without CD8+ cells
Detection of Borna disease virus genome in normal human brain tissue.
Chromosome aberrations in peripheral blood mononuclear cells infected with himan immunodeficiency virus type 1(HIV-1).
Does human immunodeficiency virus type-1 integrate in chromosomes randamly?
Enhancement of human immunodeficiency virus type 1 infectivity by Nef is producer cell-dependent.
Producer cell-dependent requirement of the Nef protein for efficient entry of HIV-1 into cells.
In vivo induction of human immunodeficiency virus type 1-specific cytotoxic T lymphocytes and delayed-type hypersensitivity by a @@S ' 23-amino acid peptide from the highly conserved region in major @@S ' core protein p24
The carboxyl-terminal region of HIV-1 Nef protein is a cell surface domain that can interact with CD4+ T cells
A novel acyl-CoA thioesterase enhances its enzymatic activity by direct binding with HIV Nef.
Growth ability of auxiliary gene mutants of human immunodeficiency virus type 1 and 2 in unstimulated peripheral blood mononuclear cells.
Multiple effects of CD4 CDR3-related peptide derivatives showing anti-HIV-1 activity on HIV-1 gp120 functions
Are defective, HIV protease-deficient particles the real culprit in AIDS?A specific type of defective particle produced after @@S ' persistent infection of lymphoid cells may be the trigger in @@S ' pathogenesis.
The morphology of the immature HIV-1 virion.
Poly(ADP-ribose) polymerase activity in various U937 cell subclones with different susceptibility to HIV-1 infection : Its dramatic decre ase following persistent virus infection.
AIDS pathogenesis: the role of accessory gene mutations, leading to formation of long-lived persistently infected cells and/or apoptosis- inducing HIV-1 particles.
The life cycle of human immunodeficiency virus type 1

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