The PPARγ Agonist Protects Cardiomyocytes from Oxidative Stress and Apoptosis via Thioredoxin Overexpression
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概要
- 論文の詳細を見る
- 2012-12-23
著者
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Cho Myeong-chan
Department Of Cardiology Chungbuk National University Hospital
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Kim Dong-woon
Department Of Cardiology Chungbuk National University Hospital
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Bae Jang-whan
Department Of Internal Medicine Chungbuk National University Hospital
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KIM Yeon-Jung
Department of Internal Medicine, College of Medicine, Chungbuk National University
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HWANG Kyung-Kuk
Department of Internal Medicine, College of Medicine, Chungbuk National University
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Hwang Kyung-kuk
Department Of Internal Medicine College Of Medicine Chungbuk National University
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Shim Tae-jin
Department Of Internal Medicine College Of Medicine Chungbuk National University
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BAE Jang-Whan
Department of Internal Medicine, College of Medicine, Chungbuk National University
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Cho Myeong-Chan
Department of Internal Medicine, College of Medicine, Chungbuk National University
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SONG Joong-Ki
Department of Internal Medicine, College of Medicine, Chungbuk National University
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PARK Keon-Jea
Department of Internal Medicine, College of Medicine, Chungbuk National University
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ISLAM Kazi
Center for Translational Medicine, Temple University School of Medicine
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KIM Sang-Min
Department of Internal Medicine, College of Medicine, Chungbuk National University
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LEE Sang-Yeub
Department of Internal Medicine, College of Medicine, Chungbuk National University
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RYU Keun
Database and Bioinformatics Laboratory, Chungbuk National University
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KIM Dong-Woon
Department of Internal Medicine, College of Medicine, Chungbuk National University
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- The PPARγ Agonist Protects Cardiomyocytes from Oxidative Stress and Apoptosis via Thioredoxin Overexpression