Overlapping DSPP Mutations Cause Dentin Dysplasia and Dentinogenesis Imperfecta
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概要
- 論文の詳細を見る
- 2008-12-01
著者
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Hart P.
Nhgri Nih
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Simmer J.
Department Of Biologic And Materials Science University Of Michigan School Of Dentistry Dental Resea
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Simmer J.
Department Of Biologic And Materials Sciences University Of Michigan School Of Dentistry Dental Rese
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FISHER L.
Craniofacial and Skeletal Diseases Branch
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Mcknight D.
Craniofacial And Skeletal Diseases Branch Nidcr Nih Dhhs
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HART T.
Section of Human and Craniofacial Genetics, NIDCR, NIH, DHHS
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Hart T.
Section Of Human And Craniofacial Genetics Nidcr Nih Dhhs
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Hart P.
Nhgri Nih Dhhs
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Fisher L.
Craniofacial And Skeletal Diseases Branch Nidcr Nih Dhhs
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- Hereditary Dentin Defects
- Fam83h is Associated with Intracellular Vesicles and ADHCAI
- Stem Cell Properties of Human Dental Pulp Stem Cells
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- Amelogenin p. M1T and p. W4S Mutations Underlying Hypoplastic X-linked Amelogenesis Imperfecta
- Mmp-20 and Klk4 Cleavage Site Preferences for Amelogenin Sequences
- DPPI May Activate KLK4 during Enamel Formation
- Cleavage Site Specificity of MMP-20 for Secretory-stage Ameloblastin
- Splicing Determines the Glycosylation State of Ameloblastin
- Comparison of Stem-cell-mediated Osteogenesis and Dentinogenesis
- Overlapping DSPP Mutations Cause Dentin Dysplasia and Dentinogenesis Imperfecta
- Altered Enamelin Phosphorylation Site Causes Amelogenesis Imperfecta
- ENAM Mutations in Autosomal-dominant Amelogenesis Imperfecta
- Novel WDR72 Mutation and Cytoplasmic Localization
- Regulation of Dental Enamel Shape and Hardness
- Transgenic Rescue of Enamel Phenotype in Ambn Null Mice
- Novel MSX1 Frameshift Causes Autosomal-dominant Oligodontia
- FAM83H Mutations Cause ADHCAI and Alter Intracellular Protein Localization